Secondary Pulmonary Edema

R. Parker
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引用次数: 0

Abstract

Secondary pulmonary edema may result from mitral stenosis, mitral regurgitation, or left heart failure. Each of these pathologic conditions elevate left atrial pressure and thereby increase pulmonary microvascular pressure. However, the degree to which left atrial pressure is elevated will determine if intraalveolar edema will occur. The rate at which fluid will traverse the pulmonary microvascular membrane is dependent upon the balance of intravascular and extravascular hydrostatic pressures, the intravascular to extravascular oncotic pressure gradient, hydraulic conductivity of the pulmonary microvascular membrane, and the permeability of the pulmonary microvascular membrane to plasma proteins. The relationship of these pressures and characteristics of the pulmonary microvascular membrane can be described by the Starling Equation:
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继发性肺水肿
继发性肺水肿可由二尖瓣狭窄、二尖瓣反流或左心衰引起。每一种病理情况都会升高左心房压,从而增加肺微血管压。然而,左房压升高的程度将决定是否会发生肺泡内水肿。流体通过肺微血管膜的速率取决于血管内和血管外静水压力的平衡、血管内和血管外的肿瘤压力梯度、肺微血管膜的水力传导率以及肺微血管膜对血浆蛋白的渗透性。这些压力与肺微血管膜特性的关系可以用Starling方程来描述:
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