Correction to “Inflammatory Response and Endothelial Dysfunction in the Hearts of Mice Co-Exposed to SO2, NO2, and PM2.5”

IF 3.2 3区 医学 Q2 ENVIRONMENTAL SCIENCES Environmental Toxicology Pub Date : 2024-10-29 DOI:10.1002/tox.24432
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Abstract

Zhang, Y., Ji, X., Ku, T. and Sang, N. (2016), Inflammatory response and endothelial dysfunction in the hearts of mice co-exposed to SO2, NO2, and PM2.5 . Environmental Toxicology, 31: 19962005. https://doi.org/10.1002/tox.22200.

In Figure 4 of the original paper, there was a misuse of the picture that occurred in the low-dose group (A2), and the corrected Figure 4 is presented here.

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更正 "共同暴露于二氧化硫、二氧化氮和 PM2.5 的小鼠心脏的炎症反应和内皮功能障碍"。
张艳,纪晓明,顾涛,桑宁(2016),SO2、NO2和PM2.5共同暴露小鼠心脏炎症反应和内皮功能障碍。环境毒理学杂志,31(1):1996-2005。https://doi.org/10.1002/tox.22200。在原论文的图4中,出现了低剂量组(A2)对图片的误用,这里给出了更正后的图4。
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来源期刊
Environmental Toxicology
Environmental Toxicology 环境科学-毒理学
CiteScore
7.10
自引率
8.90%
发文量
261
审稿时长
4.5 months
期刊介绍: The journal publishes in the areas of toxicity and toxicology of environmental pollutants in air, dust, sediment, soil and water, and natural toxins in the environment.Of particular interest are: Toxic or biologically disruptive impacts of anthropogenic chemicals such as pharmaceuticals, industrial organics, agricultural chemicals, and by-products such as chlorinated compounds from water disinfection and waste incineration; Natural toxins and their impacts; Biotransformation and metabolism of toxigenic compounds, food chains for toxin accumulation or biodegradation; Assays of toxicity, endocrine disruption, mutagenicity, carcinogenicity, ecosystem impact and health hazard; Environmental and public health risk assessment, environmental guidelines, environmental policy for toxicants.
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