Investigation of Cause of Mass Fish Death in October 2007 at Minami-Sotobori in Osaka-Jo Park

Akira Oshima, M. Kitano, Jyoji Fukuyama
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Abstract

A mass fish death occurred at Minami-Sotobori (moat) in Osaka-Jo Park on Oct. 4, 2007. It was reported that more than a thousand fish died. Features of the mass death were: 1) it involved many of the fish species that live in the moat, 2) most were fish with long body length and died with open mouth, 3) dissolved oxygen was 3.1 mg·L-1 or less throughout the water column. It was judged that the fish had died of anoxia. The air temperature had decreased rapidly seven days before the fatal accident and low temperatures continued for four days. As a result, it is thought that the epilimnion was cooled strongly and sank to the bottom during this period and that the hypolimnion, which contains less oxygen, welled up. Since the hypolimnion seems to become anaerobic under organic loading for long periods, it is possible that similar accidents will happen again when water stratification breaks down for some reason.
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2007年10月大阪jo公园Minami-Sotobori鱼类大量死亡原因调查
2007年10月4日,在大阪jo公园的南索托保里(护城河)发生了大规模的鱼类死亡事件。据报道,有一千多条鱼死亡。大量死亡的特征是:1)涉及到许多生活在护城河中的鱼类,2)大多数是长体长的鱼,张开嘴死亡,3)整个水柱的溶解氧为3.1 mg·L-1或以下。人们断定那条鱼死于缺氧。在致命事故发生前7天,气温迅速下降,低温持续了4天。因此,有人认为,在这段时间里,咸水离子被强烈冷却并沉入海底,而含氧量较少的咸水离子则涌出。由于低聚阴离子在长时间的有机负荷下似乎会变成厌氧,所以当水分层因某种原因而破裂时,类似的事故可能会再次发生。
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