Atl(atlastin)通过改变溶酶体网络调节果蝇肌肉中的mTor信号转导和自噬。

IF 14.6 1区 生物学 Q1 CELL BIOLOGY Autophagy Pub Date : 2024-01-01 Epub Date: 2023-08-30 DOI:10.1080/15548627.2023.2249794
Saurabh Srivastav, Kevin van der Graaf, Pratibha Singh, Alloysius Budi Utama, Matthew D Meyer, James A McNew, Michael Stern
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引用次数: 0

摘要

缩写:atl atlastin;ALR autophagic lysosome reformation;ER endoplasm reticulum;GFP 绿色荧光蛋白;HSP hereditary spastic paraplegia;Lamp1 lysosomal associated membrane protein 1 PolyUB polyubiquitin;RFP red fluorescent protein;spinster;mTor mechanistic Target of rapamycin;VCP valosin containing protein。
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Atl (atlastin) regulates mTor signaling and autophagy in Drosophila muscle through alteration of the lysosomal network.

Abbreviations: atl atlastin; ALR autophagic lysosome reformation; ER endoplasmic reticulum; GFP green fluorescent protein; HSP hereditary spastic paraplegia; Lamp1 lysosomal associated membrane protein 1 PolyUB polyubiquitin; RFP red fluorescent protein; spin spinster; mTor mechanistic Target of rapamycin; VCP valosin containing protein.

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来源期刊
Autophagy
Autophagy 生物-细胞生物学
CiteScore
21.30
自引率
2.30%
发文量
277
审稿时长
1 months
期刊介绍: Autophagy is a peer-reviewed journal that publishes research on autophagic processes, including the lysosome/vacuole dependent degradation of intracellular material. It aims to be the premier journal in the field and covers various connections between autophagy and human health and disease, such as cancer, neurodegeneration, aging, diabetes, myopathies, and heart disease. Autophagy is interested in all experimental systems, from yeast to human. Suggestions for specialized topics are welcome. The journal accepts the following types of articles: Original research, Reviews, Technical papers, Brief Reports, Addenda, Letters to the Editor, Commentaries and Views, and Articles on science and art. Autophagy is abstracted/indexed in Adis International Ltd (Reactions Weekly), EBSCOhost (Biological Abstracts), Elsevier BV (EMBASE and Scopus), PubMed, Biological Abstracts, Science Citation Index Expanded, Web of Science, and MEDLINE.
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