{"title":"脑脊髓炎病毒结构蛋白 VP3 与 MAVS 相互作用并促进其自噬降解,从而干扰 I 型干扰素信号通路","authors":"Xu Zhao, Zhengyang Hou, Yaxin Zhang, Daoqin Mao, Zhenfang Yan, Shunyu Yang, Jingying Xie, Ruofei Feng","doi":"10.31083/j.fbl2907273","DOIUrl":null,"url":null,"abstract":"","PeriodicalId":503756,"journal":{"name":"Frontiers in Bioscience-Landmark","volume":"30 15","pages":""},"PeriodicalIF":0.0000,"publicationDate":"2024-07-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Encephalomyocarditis Virus Structural Protein VP3 Interacts with MAVS and Promotes its Autophagic Degradation to Interfere with the Type I Interferon Signaling Pathway\",\"authors\":\"Xu Zhao, Zhengyang Hou, Yaxin Zhang, Daoqin Mao, Zhenfang Yan, Shunyu Yang, Jingying Xie, Ruofei Feng\",\"doi\":\"10.31083/j.fbl2907273\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"\",\"PeriodicalId\":503756,\"journal\":{\"name\":\"Frontiers in Bioscience-Landmark\",\"volume\":\"30 15\",\"pages\":\"\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2024-07-25\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Frontiers in Bioscience-Landmark\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.31083/j.fbl2907273\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Frontiers in Bioscience-Landmark","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.31083/j.fbl2907273","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Encephalomyocarditis Virus Structural Protein VP3 Interacts with MAVS and Promotes its Autophagic Degradation to Interfere with the Type I Interferon Signaling Pathway