纠正 "内质网伴侣蛋白 GRP78 通过调节 IRE1-TRAF2-JNK 通路参与氟化物诱导的 LS8 细胞自噬"。

IF 4.4 3区 医学 Q2 ENVIRONMENTAL SCIENCES Environmental Toxicology Pub Date : 2024-08-27 DOI:10.1002/tox.24402
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引用次数: 0

摘要

赵丽丽,王丽丽,王红华,等,“内质网伴侣蛋白GRP78通过调控IRE1-TRAF2-JNK通路参与氟化物诱导的LS8细胞自噬,”环境毒理学38,no. 5。7(2023): 1756-1767。我们发现图2 a和e中Beclin1的Western Blot (WB)图像存在重复。通过检查我们的实验数据,我们发现错误发生在复制图片的过程中。然而,我们已经反复检查了我们的结果,统计数据是准确的。只有图像复制过程有错误。现将图2 A、E、I面板中Beclin1的WB图像的三个实验结果,以及校正后的结果提供给您审阅。修改后
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Correction to “Endoplasmic Reticulum Chaperone GRP78 Participates in Fluoride-Induced Autophagy in LS8 Cells by Regulating the IRE1-TRAF2-JNK Pathway”

L. Zhao, L. Wang, H. Wang, et al., “Endoplasmic Reticulum Chaperone GRP78 Participates in Fluoride-Induced Autophagy in LS8 Cells by Regulating the IRE1-TRAF2-JNK Pathway,” Environmental Toxicology 38, no. 7 (2023): 1756–1767.

We have discovered that there is a duplication of the Western Blot (WB) images of Beclin1 in Figure 2, panels A and E. Upon reviewing our experimental data, we found that the error occurred during the copying pictures process. However, we have cross-checked our results, and the statistical data is accurate. Only the image copying process has errors. Now we provide the three experimental results of WB images of Beclin1 from Figure 2, panels A, E, and I, along with the corrected results, for your review.

After modification

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来源期刊
Environmental Toxicology
Environmental Toxicology 环境科学-毒理学
CiteScore
7.10
自引率
8.90%
发文量
261
审稿时长
4.5 months
期刊介绍: The journal publishes in the areas of toxicity and toxicology of environmental pollutants in air, dust, sediment, soil and water, and natural toxins in the environment.Of particular interest are: Toxic or biologically disruptive impacts of anthropogenic chemicals such as pharmaceuticals, industrial organics, agricultural chemicals, and by-products such as chlorinated compounds from water disinfection and waste incineration; Natural toxins and their impacts; Biotransformation and metabolism of toxigenic compounds, food chains for toxin accumulation or biodegradation; Assays of toxicity, endocrine disruption, mutagenicity, carcinogenicity, ecosystem impact and health hazard; Environmental and public health risk assessment, environmental guidelines, environmental policy for toxicants.
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