mESC 中多能到全能状态的转变通过 DUX 诱导的 DNA 复制压力激活了内在凋亡途径。

IF 6.2 2区 生物学 Q1 BIOCHEMISTRY & MOLECULAR BIOLOGY Cellular and Molecular Life Sciences Pub Date : 2024-10-26 DOI:10.1007/s00018-024-05465-z
Shunze Jia, Xinpeng Wen, Minwei Zhu, Xudong Fu
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引用次数: 0

摘要

多能小鼠胚胎干细胞(mESCs)可以过渡到类全能状态,而转录因子DUX是这种过渡的主调控因子之一。耐人寻味的是,mESCs的这种转变伴随着大量的细胞死亡,这极大地阻碍了全能细胞在体外的建立和维持,然而这种细胞死亡的潜在机制在很大程度上仍然难以捉摸。在这项研究中,我们发现 mESCs 的全能性转变会通过 DUX 的上调引发细胞死亡。具体来说,DUX诱导后会积累R环,进而导致mESCs中的DNA复制应激(RS)。这种RS会进一步激活p53和PMAIP1,最终导致依赖Caspase-9/7的内在凋亡。值得注意的是,抑制这种内在凋亡不仅能缓解细胞死亡,还能提高 mESC 的全能性转变效率。因此,我们的研究结果阐明了mESCs全能性转变过程中细胞凋亡的机制之一,并为优化体外全能性细胞的建立和维持提供了一种策略。
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The pluripotent-to-totipotent state transition in mESCs activates the intrinsic apoptotic pathway through DUX-induced DNA replication stress.

The pluripotent mouse embryonic stem cell (mESCs) can transit into the totipotent-like state, and the transcription factor DUX is one of the master regulators of this transition. Intriguingly, this transition in mESCs is accompanied by massive cell death, which significantly impedes the establishment and maintenance of totipotent cells in vitro, yet the underlying mechanisms of this cell death remain largely elusive. In this study, we found that the totipotency transition in mESCs triggered cell death through the upregulation of DUX. Specifically, R-loops are accumulated upon DUX induction, which subsequently lead to DNA replication stress (RS) in mESCs. This RS further activates p53 and PMAIP1, ultimately leading to Caspase-9/7-dependent intrinsic apoptosis. Notably, inhibiting this intrinsic apoptosis not only mitigates cell death but also enhances the efficiency of the totipotency transition in mESCs. Our findings thus elucidate one of the mechanisms underlying cell apoptosis during the totipotency transition in mESCs and provide a strategy for optimizing the establishment and maintenance of totipotent cells in vitro.

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来源期刊
Cellular and Molecular Life Sciences
Cellular and Molecular Life Sciences 生物-生化与分子生物学
CiteScore
13.20
自引率
1.20%
发文量
546
审稿时长
1.0 months
期刊介绍: Journal Name: Cellular and Molecular Life Sciences (CMLS) Location: Basel, Switzerland Focus: Multidisciplinary journal Publishes research articles, reviews, multi-author reviews, and visions & reflections articles Coverage: Latest aspects of biological and biomedical research Areas include: Biochemistry and molecular biology Cell biology Molecular and cellular aspects of biomedicine Neuroscience Pharmacology Immunology Additional Features: Welcomes comments on any article published in CMLS Accepts suggestions for topics to be covered
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