返回：长非编码 RNA ZNF667-AS1 通过抵消 microRNA-93-3p 依赖性 PEG3 的下调作用，减少宫颈癌的肿瘤侵袭和转移。

IF 6.6 2区医学 Q1 Biochemistry, Genetics and Molecular Biology Molecular Oncology Pub Date : 2024-11-27 DOI:10.1002/1878-0261.13777

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摘要

撤回：Li Y-J, Yang Z, Wang Y-Y, Wang Y.长非编码RNA ZNF667-AS1通过对抗microRNA-93-3p依赖的PEG3下调减少宫颈癌的肿瘤侵袭和转移Mol Oncol.2019;13(11):2375-2392. https://doi.org/10.1002/1878-0261.12565 上述文章于2019年10月17日在线发表于Wiley Online Library (wileyonlinelibrary.com)，经期刊主编Kevin Ryan、FEBS Press和John Wiley & Sons Ltd.协议撤回。在对第三方提出的疑虑进行调查后，作者同意撤稿。第三方提出的疑虑揭示了难以置信的 Western 印迹数据（图 5B,L,G,Q）和图 6B 中的图像重复。由于作者未能提供原始数据，编辑对所提供的数据失去了信心。因此，编辑认为该论文的结论大打折扣，并撤回该论文。

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RETRACTION: Long noncoding RNA ZNF667-AS1 reduces tumor invasion and metastasis in cervical cancer by counteracting microRNA-93-3p-dependent PEG3 downregulation.

Retraction: Li Y-J, Yang Z, Wang Y-Y, Wang Y. Long noncoding RNA ZNF667-AS1 reduces tumor invasion and metastasis in cervical cancer by counteracting microRNA-93-3p-dependent PEG3 downregulation. Mol Oncol. 2019;13(11):2375-2392. https://doi.org/10.1002/1878-0261.12565 The above article, published online on 17 October 2019 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the journal Editor-in-Chief, Kevin Ryan; FEBS Press; and John Wiley & Sons Ltd. The retraction has been agreed upon following an investigation into concerns raised by a third party, which revealed implausible Western blot data (Fig. 5B,L,G,Q), and an image duplication in Fig. 6B. The authors' failure to respond with the original raw data has led the editors to lose confidence in the data presented. Therefore, the editors consider the conclusions substantially compromised and are retracting the paper.

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来源期刊

Molecular Oncology Biochemistry, Genetics and Molecular Biology-Molecular Medicine

CiteScore

11.80

自引率

1.50%

发文量

203

审稿时长

10 weeks

期刊介绍： Molecular Oncology highlights new discoveries, approaches, and technical developments, in basic, clinical and discovery-driven translational cancer research. It publishes research articles, reviews (by invitation only), and timely science policy articles. The journal is now fully Open Access with all articles published over the past 10 years freely available.