肠套叠继发于肿块和摄入

María Inguanzo-Ortiz
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摘要

我们报告了一个先前健康的9个月大的婴儿,由于7小时的阵发性腹痛和植物性症状的演变,交替出现活动不足和进行性嗜睡,他去了急诊科。伴有厌食和呕吐,无发热,最后一次大便在12小时前,特征正常。生命体征正常。在体格检查中,一般状态与肿胀的关系值得注意。脑膜征为阴性,无神经病灶。腹部疼痛,右侧有压痛,无腹胀迹象。血液分析正常,尿液毒素检测呈阴性。腹部x线片显示小肠和大肠内有大量不透射线的物质,未见肠穿孔或肺积病征象(图1)。腹部超声检查证实回肠结肠内陷。父母否认服用了对比剂,但在症状出现前10小时报告了大量的沙子摄入。针对临床怀疑大量进沙后继发肠内陷,采用超声引导下静液复位。在手术过程中,症状突然消失,患者无症状。大量的沙子被移走了。由于摄入沙子而引起的沙性肠病在马中相当常见食土癖是异食癖的一种形式,在某些文化中被认为是正常的。其病因是多因素的,被认为可能是微量营养素缺乏的标志其并发症之一是肠梗阻,可能是肠套叠的起源点
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Intestinal intussusception secondary to massives and intake
We present the case of a previously healthy 9-month-old infant who went to the Emergency Department due to a 7-hour evolution of paroxysms of abdominal pain with vegetative symptoms that alternated with hypoactivity and progressive lethargy. Anorexia and vomiting were also associated, fever was absent and the last stool was 12 hours prior, with normal characteristics. Vital signs were normal. On physical examination, the involvement of the general state with obtundation was noteworthy. The meningeal signs were negative, and there was no neurological focality. The abdomen was painful and presented tenderness on the right flank without signs of peritonism. Blood analysis was normal, and urine was negative for toxins. Abdominal radiograph revealed abundant radiopaque material in the small and large intestines without signs of intestinal perforation or pneumatosis (Figure 1). Abdominal ultrasonography confirmed ileocolic invagination. The parents denied the administration of contrast but reported copious sand intake 10 hours before the onset of symptoms. In view of the clinical suspicion of intestinal invagination secondary to massive sand intake, ultrasound guided hydrostatic reduction was performed. During the procedure, the symptoms suddenly ceased and the patient became asymptomatic. A large amount of sand was removed. Sand enteropathy due to ingestion of sand is rather common in horses.1 Geophagia is a form of pica considered normal in some cultures. Its aetiology is multifactorial and has been postulated as a marker of possible micronutrient deficit.2 One of its complications is intestinal obstruction and may be the point of origin of an intestinal intussusception.3-5
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