通过FODMAP-diet和Rebamipid成功Diagnosтic治疗膜消化受损的肠病

E. A. Kondrashova, N. Belostotsky, A. Strachuk, E. Baulo, A. Parfenov
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引用次数: 0

摘要

十二指肠黏膜中双糖酶活性的降低使得有可能在K.肠病患者中建立膜消化受损(EIMD)。在EIMD期间,回肠远端双糖酶活性的增加是根据近端梯度类型形成代偿反应的验证。用细胞保护剂利巴米胺治疗12周可增加EIMD患者小肠的酶活性。
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Successful Diagnosтic and Treatment of Enteropathy with Impaired Membrane Digestion by FODMAP-diet and Rebamipid
A decrease in the activity of disaccharidases in the duodenal mucosa made it possible to establish in patient K. enteropathy with impaired membrane digestion (EIMD). An increase in the activity of disaccharidases in the distal ileum during EIMD is a verification of the formation of a compensatory reaction according to the type of proximodistal gradients. Treatment with the cytoprotector rebamipide for 12 weeks increases the enzymatic activity of the small intestine in EIMD.
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