Correction: WDFY3 mutation alters laminar position and morphology of cortical neurons.

IF 6.3 1区医学 Q1 GENETICS & HEREDITY Molecular Autism Pub Date : 2023-01-31 DOI:10.1186/s13229-023-00539-4

Zachary A Schaaf, Lyvin Tat, Noemi Cannizzaro, Alexios A Panoutsopoulos, Ralph Green, Thomas Rülicke, Simon Hippenmeyer, Konstantinos S Zarbalis

引用次数: 0

查看原文

微信好友朋友圈 QQ好友复制链接

本刊更多论文

更正:WDFY3突变改变了皮层神经元的层状位置和形态。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

求助全文

约1分钟内获得全文去求助

来源期刊

Molecular Autism GENETICS & HEREDITY-NEUROSCIENCES

CiteScore

12.10

自引率

1.60%

发文量

审稿时长

17 weeks

期刊介绍： Molecular Autism is a peer-reviewed, open access journal that publishes high-quality basic, translational and clinical research that has relevance to the etiology, pathobiology, or treatment of autism and related neurodevelopmental conditions. Research that includes integration across levels is encouraged. Molecular Autism publishes empirical studies, reviews, and brief communications.

期刊最新文献

Auditory N1 event-related potential amplitude is predictive of serum concentration of BPN14770 in fragile X syndrome. Characterizing genetic pathways unique to autism spectrum disorder at multiple levels of biological analysis. Developmental trajectories in infants and pre-school children with Neurofibromatosis 1. Superior temporal sulcus folding, functional network connectivity, and autistic-like traits in a non-clinical population. Structure-function coupling in white matter uncovers the hypoconnectivity in autism spectrum disorder.