SEORANG PENDERITA INFARK MIOKARD AKUT TANPA ELEVASI SEGMEN ST DENGAN SINDROMA QT MEMANJANG

Ketut Angga Aditya Putra Pramana, Yusra Pintaningrum, Jatno Karjono
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Abstract

We reported a 64-year-old woman was admitted to Emergency Department of dr.Soetomo hospital with fainting and previous chest discomfort, then we diagnosed this patient with Non ST Elevation Myocard Infarct, diabetes mellitus, hypertension stage II, and long QT syndrome (LQTS) with torsades de pointes episodes.  There are two important tests to determine LQTS, electrocardiogram (ECG) and a genetic test. Two kinds of LQTS, congenital LQTS is caused by mutations in genes coding, and acquired LQTS often is associated with drugs or metabolit abnormalities.  Myocardial ischemia could change QT interval regionally in the area of ischemia, and these changes are related to extent and severity of coronary atherosclerosis. There are two major treatment options for patients with LQTS, medications and ICD/pacemaker implantation. Beta-blockers are the drugs of choice.  
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急性心肌梗死患者t端长QT综合征,无明显升高
我们报告了一名64岁的女性因昏厥和既往胸部不适被送往soetomo医生医院急诊科,然后我们诊断该患者为非ST段抬高型心肌梗死、糖尿病、高血压II期和长QT综合征(LQTS)伴椎体扭曲发作。有两个重要的测试来确定LQTS,心电图(ECG)和基因测试。两种LQTS,先天性LQTS是由基因编码突变引起的,而获得性LQTS往往与药物或代谢异常有关。心肌缺血可使局部局部QT间期发生改变,这种改变与冠状动脉粥样硬化的程度和严重程度有关。LQTS患者有两种主要的治疗选择,药物治疗和ICD/起搏器植入。受体阻滞剂是首选药物。
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