Dissecting aneurysm of thoracic aorta: reappraisal of radiologic diagnosis.

Y Itzchak, T Rosenthal, R Adar, Z J Rubenstein, Y Lieberman, V Deutsch
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引用次数: 30

Abstract

In a series of 24 cases of acute dissecting aneurysm of the aorta (not including Marfan's disease) the diagnosis was usually suspected on the basis of the clinical picture and plain chest roentgenograms. The most consistent clinical sign was severe pain. Absent pulses and a neurological deficit were each noted in only five patients. In many cases there was no correlation between the clinical picture and the type or the extent of the dissection. Widening of the aortic arch and obliteration of the aortic knob with displacement of the trachea to the right are the most common signs in plain chest roentgenograms. A barium swallow examination in these cases reveals an elongated compression and displacement of the esophagus by the aortic arch. Calcification in the area of the aortic arch is the exception rather than the rule in dissecting aneurysms. Angiography is essential for the definitive diagnosis of dissecting aneurysms. The diagnosis is based on the demonstration of two channels, either by the presence of a linear radiolucency separating the two lumens, or by differences in flow that present as delayed opacification or delayed washout. If only the true lumen is opacified, widening of the outer extraluminal border of the aorta or narrowing of the lumen indicates the presence of a dissection. Abnormal catheter recoil and position were helpful in only two cases, and are not informative when the false lumen is catheterized. Failure to visualize main aortic branches was not always due to involvement by the dissection. It can also be caused by reduced flow due to severe proximal compression of the main lumen. The exact location of the intimal tears is usually not demonstrated unless additional injections are made in the area assumed to contain the tear. If only the false lumen is opacified in the ascending aorta, this can be recognized by the demonstration of a blind end, by failure to visualize the sinuses of Valsalva, from flattening of the medial border of the opacified channel, and from delayed washout in the blind end.

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胸主动脉夹层动脉瘤:影像学诊断的再评价。
在24例急性夹层动脉瘤(不包括马凡氏病)中,通常根据临床表现和胸部x线平片怀疑诊断。最一致的临床症状是剧烈疼痛。只有5名患者出现脉搏缺失和神经功能障碍。在许多病例中,临床表现与解剖类型或程度没有相关性。胸片平片最常见的征象是主动脉弓变宽和主动脉旋钮闭塞伴气管向右移位。这些病例的钡餐检查显示主动脉弓对食管的压迫和移位拉长。主动脉弓区域的钙化是动脉瘤解剖中的例外,而不是常规。血管造影对于解剖性动脉瘤的明确诊断至关重要。诊断是基于两个通道的表现,要么是通过将两个管腔分开的线性辐射率的存在,要么是通过表现为延迟混浊或延迟冲洗的血流差异。如果只有真腔混浊,主动脉腔外边界变宽或管腔变窄表明存在夹层。异常的导管后坐力和位置仅在两个病例中有帮助,而在假腔置管时没有帮助。不能看到主动脉主干并不总是由于夹层的累及。它也可以由严重近端压迫主腔导致的流量减少引起。除非在假定包含撕裂的区域进行额外的注射,否则通常无法确定内膜撕裂的确切位置。如果在升主动脉中只有假腔混浊,则可以通过显示盲端、无法看到Valsalva窦、混浊通道内侧边界变平以及盲端延迟洗脱来识别。
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