{"title":"[Electroencephalography and cranial computed tomography in a posterior infarct].","authors":"B Griewing, A Lütcke, L Mertins","doi":"","DOIUrl":null,"url":null,"abstract":"<p><p>In this study we interpreted the electroencephalograms of 50 patients, all suffering from a cerebral infarction occurred in the circulation area of A. cerebri posterior with or without involvement of thalamic structures. The diagnosis was established by clinical findings and via CT-examinations. Besides the EEG-findings which always occurred in the occipital region we found alterations which exceeded the above mentioned posterior disorder of the brain function. In most cases these have been bilateral paroxysmal disorders projected into the temporal regions but lateralized to the region of the existing posterior infarction. If we had CT-examinations of cerebral infarctions in the circulation area of A. cerebri posterior without an involvement of thalamic structures, there was no projected activity which exceeded the posterior disorder in EEG. In all cases with the additional affection of thalamic structures in CT the bilateral paroxysmal EEG disorders into the temporal region were found.</p>","PeriodicalId":75812,"journal":{"name":"EEG-EMG Zeitschrift fur Elektroenzephalographie, Elektromyographie und verwandte Gebiete","volume":"23 3","pages":"147-50"},"PeriodicalIF":0.0000,"publicationDate":"1992-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"EEG-EMG Zeitschrift fur Elektroenzephalographie, Elektromyographie und verwandte Gebiete","FirstCategoryId":"1085","ListUrlMain":"","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
引用次数: 0
Abstract
In this study we interpreted the electroencephalograms of 50 patients, all suffering from a cerebral infarction occurred in the circulation area of A. cerebri posterior with or without involvement of thalamic structures. The diagnosis was established by clinical findings and via CT-examinations. Besides the EEG-findings which always occurred in the occipital region we found alterations which exceeded the above mentioned posterior disorder of the brain function. In most cases these have been bilateral paroxysmal disorders projected into the temporal regions but lateralized to the region of the existing posterior infarction. If we had CT-examinations of cerebral infarctions in the circulation area of A. cerebri posterior without an involvement of thalamic structures, there was no projected activity which exceeded the posterior disorder in EEG. In all cases with the additional affection of thalamic structures in CT the bilateral paroxysmal EEG disorders into the temporal region were found.