Neuronal metabolism and sympathetic pupillary activity in infants.

L P SriVatsa
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Abstract

A delayed or immature transmission dynamics of an otherwise diffuse mesolimbic catecholamine circuit is suggested to be the reason behind lack of pupillary activity and reflex often observed in infants less than one year of age. This deficiency is known to be fully compensated in later years following a growing curve of neuronal metabolic change and as well as a curve for synaptoogenesis.

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婴儿的神经代谢和交感瞳孔活动。
在一岁以下的婴儿中经常观察到瞳孔活动和反射的缺乏,这是由于弥漫性中边缘儿茶酚胺回路的延迟或不成熟的传递动力学。这种缺陷在以后的几年里随着神经元代谢变化的增长曲线和突触发生的曲线被完全补偿。
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