Sule Gokce, Hande Dede, Mehmet Bilal Aracı, F. Koç
{"title":"Primary Active Epstein-Barr Virus Infection Coexisting With Henoch-Schonlein Purpura in a Child: A Rare Trigger Factor of Henoch-Schonlein Purpura","authors":"Sule Gokce, Hande Dede, Mehmet Bilal Aracı, F. Koç","doi":"10.33552/ACCS.2018.01.000501","DOIUrl":null,"url":null,"abstract":"Henoch-Schonlein purpura (HSP), also known as IgA vasculitis, is the most common vasculitis that effects the small vessels of the skin, joints, gastrointestinal tract, and kidneys in childhood. Epstein barr virus infection is a mild, asymptomatic infection and uncomplicated disease. It is stated that an Epstein–Barr virus infection might be a trigerr factor for Henoch-Schonlein purpura and this togetherness is rarely reported. Herein, we described a patient with Henoch-Schonlein purpura associated with primary active EBV infection. We hope our case report will create awareness of EBV in HSP vasculitis and we suggest that routine serologic investigations may help to detect the possible etiology and/or stimulant factors of HSP.","PeriodicalId":151968,"journal":{"name":"Archives of Clinical Case Studies","volume":"60 1","pages":"0"},"PeriodicalIF":0.0000,"publicationDate":"2018-08-21","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Archives of Clinical Case Studies","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.33552/ACCS.2018.01.000501","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
引用次数: 0
Abstract
Henoch-Schonlein purpura (HSP), also known as IgA vasculitis, is the most common vasculitis that effects the small vessels of the skin, joints, gastrointestinal tract, and kidneys in childhood. Epstein barr virus infection is a mild, asymptomatic infection and uncomplicated disease. It is stated that an Epstein–Barr virus infection might be a trigerr factor for Henoch-Schonlein purpura and this togetherness is rarely reported. Herein, we described a patient with Henoch-Schonlein purpura associated with primary active EBV infection. We hope our case report will create awareness of EBV in HSP vasculitis and we suggest that routine serologic investigations may help to detect the possible etiology and/or stimulant factors of HSP.