Ibrutinib attenuated DSS-induced ulcerative colitis, oxidative stress, and the inflammatory cascade by modulating the PI3K/Akt and JNK/NF-κB pathways

Liangliang Hao, Lina Alkry, Abdullah Alattar, Muhammad Faheem, Reem Alshaman, F. Shah, Shupeng Li
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引用次数: 3

Abstract

The mammalian gastrointestinal tract is continuously vulnerable to numerous bacteria and food additives as well as to environmental tox-ins that damage the epithelia of the gut [1]. Disturbance in intestinal
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伊鲁替尼通过调节PI3K/Akt和JNK/NF-κB通路减轻dss诱导的溃疡性结肠炎、氧化应激和炎症级联反应
哺乳动物的胃肠道一直很容易受到许多细菌和食品添加剂以及环境毒素的伤害,这些毒素会损害肠道上皮。肠道紊乱
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Overlap between Sjogren’s syndrome and anti-synthetase syndrome: association or coincidence? Ibrutinib attenuated DSS-induced ulcerative colitis, oxidative stress, and the inflammatory cascade by modulating the PI3K/Akt and JNK/NF-κB pathways Grisel syndrome and peripheral arthritis simultaneously occurred in a 7-year-old Chinese boy with Kawasaki disease Application of three-dimensional printing technology for spinal tuberculosis Two complications in patients with systemic lupus erythematosus: lupus cystitis and lupus enteritis
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