[Functional study in early strabismus: translucent screens and electro-nystagmography].

A Spielmann
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引用次数: 0

Abstract

The symptoms of infantile strabismus are triggered by fixation, chiefly monocular fixation. On the excluded eye, "occlusion deviations" (elevation, esodeviation, extorsion) are well observed through a "translucent" unilateral cover. These deviations are super-imposed upon a position of rest without fixation which may be an orthoposition, well observed through bilateral translucent occluders. They are caused by a rupture of binocular retinal stimuli which does not mean rupture of fusion (ex: the darkening Bielschowsky test). Electro-oculographic studies confirm the prevalence of adduction during fixation (nystagmus, pursuit, optokinetic nystagmus). This directional asymmetry is typical of the infant's immature system of fixation. In infantile strabismus, it persists because of missing binocular links (corpus callosum--cortex----midbrain) as shown by the study of the so-called congenital monophthalm syndrome (unilateral congenital blindness).

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[早期斜视的功能研究:半透明屏和眼震电图]。
婴儿斜视的症状是由固定引起的,主要是单眼固定。在被排除在外的眼睛上,通过“半透明”的单侧遮盖物可以很好地观察到“咬合偏差”(抬高、内偏、外斜)。这些偏差叠加在没有固定的休息位置上,这可能是正位,通过双侧半透明咬合器可以很好地观察到。它们是由双眼视网膜刺激破裂引起的,这并不意味着融合破裂(例如:变暗的Bielschowsky试验)。眼电图研究证实了固定时眼球内收的普遍性(眼球震颤、眼球追逐、眼动性眼球震颤)。这种方向不对称是婴儿不成熟的固定系统的典型特征。在婴儿斜视中,它持续存在是因为缺少双目联系(胼胝体-皮层----中脑),所谓的先天性单眼综合征(单侧先天性失明)的研究表明。
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