Correction to “Pharmacological Characterizations of anti-Dementia Memantine Nitrate via Neuroprotection and Vasodilation in Vitro and in Vivo”

IF 4.1 3区 医学 Q2 BIOCHEMISTRY & MOLECULAR BIOLOGY ACS Chemical Neuroscience Pub Date : 2024-04-28 DOI:10.1021/acschemneuro.4c00233
Shinghung Mak, Zheng Liu, Liangmiao Wu, Baojian Guo, Fangcheng Luo, Ziyan Liu, Shengquan Hu, Jiajun Wang, Guozhen Cui, Yewei Sun, Yuqiang Wang, Gaoxiao Zhang*, Yifan Han* and Zaijun Zhang*, 
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对 "硝酸美金刚酯通过体外和体内神经保护和血管扩张抗痴呆的药理学特征 "的更正
图3中的荧光照片显示了谷氨酸诱导的小脑颗粒神经元(CGNs)凋亡标志的Hoechst染色结果。遗憾的是,显示美金刚减弱的照片被错误地替换成了对照组的照片,而对照组的照片被使用了两次。在本研究中,美金刚仅作为 Hoechst 染色的阳性对照。由于 Hoechst 染色检测方法已经非常成熟,因此误用照片并不影响本研究的整体结论。与图 3 无关的其他实验也支持这些结论。图 3 的正确版本应该如下:图 3.MN-12 可减轻谷氨酸诱导的 CGNs 凋亡。在 8 DIV 时,用或不用 30 μM MN-12 或 3 μM MEM 预孵育 CGNs,然后在 2 小时后将其暴露于 100 μM 谷氨酸。谷氨酸挑战 24 小时后,用相差显微镜和 Hoechst 33342 染色(5 μg/mL)检测 CGNs 10 分钟,然后用荧光显微镜观察。(A) 箭头所示为显示亮蓝色核凝聚的凋亡细胞。(B) 用 Hoechst 染色法对凋亡细胞进行计数,与 A 组相同。 (C) 裂解经上述处理的 CGN,使用针对凋亡相关蛋白 bcl-2 的特异性抗体进行 Western 印迹检测。数据为三个独立实验的平均值 ± SEM。与对照组相比,##p < 0.01;与谷氨酸组相比,**p < 0.01。本文尚未被其他出版物引用。图 3.MN-12 可减轻谷氨酸诱导的 CGNs 凋亡。8 DIV时,用或不用30 μM MN-12或3 μM MEM预孵育CGNs,然后在2小时后暴露于100 μM谷氨酸。谷氨酸挑战 24 小时后,用相差显微镜和 Hoechst 33342 染色(5 μg/mL)检测 CGNs 10 分钟,然后用荧光显微镜观察。(A) 箭头所示为显示亮蓝色核凝聚的凋亡细胞。(B) 用 Hoechst 染色法对凋亡细胞进行计数,与 A 组相同。 (C) 裂解经上述处理的 CGN,使用针对凋亡相关蛋白 bcl-2 的特异性抗体进行 Western 印迹检测。数据为三个独立实验的平均值 ± SEM。与对照组相比,##p < 0.01;与谷氨酸组相比,**p < 0.01。
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来源期刊
ACS Chemical Neuroscience
ACS Chemical Neuroscience BIOCHEMISTRY & MOLECULAR BIOLOGY-CHEMISTRY, MEDICINAL
CiteScore
9.20
自引率
4.00%
发文量
323
审稿时长
1 months
期刊介绍: ACS Chemical Neuroscience publishes high-quality research articles and reviews that showcase chemical, quantitative biological, biophysical and bioengineering approaches to the understanding of the nervous system and to the development of new treatments for neurological disorders. Research in the journal focuses on aspects of chemical neurobiology and bio-neurochemistry such as the following: Neurotransmitters and receptors Neuropharmaceuticals and therapeutics Neural development—Plasticity, and degeneration Chemical, physical, and computational methods in neuroscience Neuronal diseases—basis, detection, and treatment Mechanism of aging, learning, memory and behavior Pain and sensory processing Neurotoxins Neuroscience-inspired bioengineering Development of methods in chemical neurobiology Neuroimaging agents and technologies Animal models for central nervous system diseases Behavioral research
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