RETRACTION: Lack of MicroRNA-155 Ameliorates Renal Fibrosis by Targeting PDE3A/TGF-β1/Smad Signaling in Mice with Obstructive Nephropathy

IF 3.3 3区生物学 Q3 CELL BIOLOGY Cell Biology International Pub Date : 2024-06-06 DOI:10.1002/cbin.12194

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Abstract

RETRACTION: W. Xi, X. Zhao, M. Wu, W. Jia, and H. Li, “Lack of MicroRNA-155 Ameliorates Renal Fibrosis by Targeting PDE3A/TGF-β1/Smad Signaling in Mice with Obstructive Nephropathy”, Cell Biology International 42, no. 11 (2018): 1523-1532. https://doi.org/10.1002/cbin.11038

The above article, published online on 6 August 2018 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the journal Editor-in-Chief, Sergio Schenkman; the International Federation for Cell Biology; and John Wiley & Sons Ltd.

The retraction has been agreed due to concerns raised by third parties on the data presented in the article. Several flaws and inconsistencies between results presented and experimental methods described were found. Furthermore, several image elements in Figure 2 A were found to have been published previously in a different scientific context. Thus, the editors consider the conclusions of this article to be invalid. The authors have been informed of the decision of retraction but were not available for a final confirmation.

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返回：通过靶向阻塞性肾病小鼠的 PDE3A/TGF-β1/Smad 信号，缺乏 MicroRNA-155 可改善肾脏纤维化。

撤回：W. Xi, X. Zhao, M. Wu, W. Jia, and H. Li, "Lack of MicroRNA-155 Ameliorates Renal Fibrosis by Targeting PDE3A/TGF-β1/Smad Signaling in Mice with Obstructive Nephropathy", Cell Biology International 42, no：1523-1532。https://doi.org/10.1002/cbin.11038 上述文章于 2018 年 8 月 6 日在线发表于 Wiley Online Library (wileyonlinelibrary.com)，经期刊主编 Sergio Schenkman、国际细胞生物学联合会和 John Wiley & Sons Ltd.同意，已被撤回。之所以同意撤稿，是因为第三方对文章中提供的数据表示担忧。我们发现，文章中介绍的结果与实验方法之间存在若干缺陷和不一致之处。此外，还发现图 2 A 中的几个图像元素以前曾在不同的科学背景下发表过。因此，编辑认为这篇文章的结论无效。作者已被告知撤稿决定，但无法得到最终确认。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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来源期刊

Cell Biology International 生物-细胞生物学

CiteScore

7.60

自引率

0.00%

发文量

208

审稿时长

1 months

期刊介绍： Each month, the journal publishes easy-to-assimilate, up-to-the minute reports of experimental findings by researchers using a wide range of the latest techniques. Promoting the aims of cell biologists worldwide, papers reporting on structure and function - especially where they relate to the physiology of the whole cell - are strongly encouraged. Molecular biology is welcome, as long as articles report findings that are seen in the wider context of cell biology. In covering all areas of the cell, the journal is both appealing and accessible to a broad audience. Authors whose papers do not appeal to cell biologists in general because their topic is too specialized (e.g. infectious microbes, protozoology) are recommended to send them to more relevant journals. Papers reporting whole animal studies or work more suited to a medical journal, e.g. histopathological studies or clinical immunology, are unlikely to be accepted, unless they are fully focused on some important cellular aspect. These last remarks extend particularly to papers on cancer. Unless firmly based on some deeper cellular or molecular biological principle, papers that are highly specialized in this field, with limited appeal to cell biologists at large, should be directed towards journals devoted to cancer, there being very many from which to choose.