MAGE-A4-Responsive Plasma Cells Promote Non-Small Cell Lung Cancer

bioRxiv Pub Date : 2024-07-16 DOI:10.1101/2024.07.10.602985
Dominique Armstrong, Cheng-Yen Chang, Monica J. Hong, Linda Green, William Hudson, Yichao Shen, Li-Zhen Song, Sheetal Jammi, Benjamin Casal, Chad J. Creighton, Alexandre Carisey, X. Zhang, Neil J. McKenna, Sung Wook Kang, Hyun-Sung Lee, D. Corry, F. Kheradmand
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Abstract

Adaptive immunity is critical to eliminate malignant cells, while multiple tumor-intrinsic factors can alter this protective function. Melanoma antigen-A4 (MAGE-A4), a cancer-testis antigen, is expressed in several solid tumors and correlates with poor survival in non-small cell lung cancer (NSCLC), but its role in altering antitumor immunity remains unclear. We found that expression of MAGE-A4 was highly associated with the loss of PTEN, a tumor suppressor, in human NSCLC. Here we show that constitutive expression of human MAGE-A4 combined with the loss of Pten in mouse airway epithelial cells results in metastatic adenocarcinoma enriched in CD138+ CXCR4+ plasma cells, predominantly expressing IgA. Consistently, human NSCLC expressing MAGE-A4 showed increased CD138+ IgA+ plasma cell density surrounding tumors. The abrogation of MAGE-A4-responsive plasma cells (MARPs) decreased tumor burden, increased T cell infiltration and activation, and reduced CD163+CD206+ macrophages in mouse lungs. These findings suggest MAGE-A4 promotes NSCLC tumorigenesis, in part, through the recruitment and retention of IgA+ MARPs in the lungs.
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MAGE-A4反应性浆细胞促进非小细胞肺癌的发生
适应性免疫对于消灭恶性细胞至关重要,而多种肿瘤内在因素会改变这种保护功能。黑色素瘤抗原-A4(MAGE-A4)是一种癌睾丸抗原,在多种实体瘤中都有表达,并与非小细胞肺癌(NSCLC)的不良生存率相关,但它在改变抗肿瘤免疫中的作用仍不清楚。我们发现,在人类 NSCLC 中,MAGE-A4 的表达与肿瘤抑制因子 PTEN 的缺失高度相关。在这里,我们发现人类 MAGE-A4 的组成型表达与小鼠气道上皮细胞中 Pten 的缺失相结合,会导致转移性腺癌,其中富含 CD138+ CXCR4+ 浆细胞,主要表达 IgA。同样,表达 MAGE-A4 的人类 NSCLC 显示肿瘤周围 CD138+ IgA+ 浆细胞密度增加。消减 MAGE-A4 反应性浆细胞(MARPs)可减少肿瘤负荷,增加 T 细胞浸润和活化,并减少小鼠肺部 CD163+CD206+ 巨噬细胞。这些发现表明,MAGE-A4 部分是通过在肺部招募和保留 IgA+ MARPs 来促进 NSCLC 肿瘤发生的。
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