Emerging and novel technologies in reproductive and developmental toxicology

IF 2.8 4区 医学 Q2 REPRODUCTIVE BIOLOGY Reproductive toxicology Pub Date : 2024-08-31 DOI:10.1016/j.reprotox.2024.108705
Susan E. Maier, Robert M. Cabrera, Yichang Chen, Neil Vargesson
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生殖与发育毒理学中的新兴技术。
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来源期刊
Reproductive toxicology
Reproductive toxicology 生物-毒理学
CiteScore
6.50
自引率
3.00%
发文量
131
审稿时长
45 days
期刊介绍: Drawing from a large number of disciplines, Reproductive Toxicology publishes timely, original research on the influence of chemical and physical agents on reproduction. Written by and for obstetricians, pediatricians, embryologists, teratologists, geneticists, toxicologists, andrologists, and others interested in detecting potential reproductive hazards, the journal is a forum for communication among researchers and practitioners. Articles focus on the application of in vitro, animal and clinical research to the practice of clinical medicine. All aspects of reproduction are within the scope of Reproductive Toxicology, including the formation and maturation of male and female gametes, sexual function, the events surrounding the fusion of gametes and the development of the fertilized ovum, nourishment and transport of the conceptus within the genital tract, implantation, embryogenesis, intrauterine growth, placentation and placental function, parturition, lactation and neonatal survival. Adverse reproductive effects in males will be considered as significant as adverse effects occurring in females. To provide a balanced presentation of approaches, equal emphasis will be given to clinical and animal or in vitro work. Typical end points that will be studied by contributors include infertility, sexual dysfunction, spontaneous abortion, malformations, abnormal histogenesis, stillbirth, intrauterine growth retardation, prematurity, behavioral abnormalities, and perinatal mortality.
期刊最新文献
Corrigendum to "Intergenerational sex-specific transcriptomic alterations in gonads of mice after parental lead exposure" [Reprod. Toxicol. 140 (2026) 109154]. Salidroside Attenuates Busulfan-Induced Testicular Injured Via Activation of The Nrf2/GPX4 Axis In Mice. Artificial Sweeteners and Male Infertility: Network Toxicology and Experimental Evidence Reveal FGFR1 as the Critical Target. Differential disruption of gonadal development by DEHP and paracetamol in male and female Wistar rats. Editorial: Reproductive and developmental effects of micro- and nano-plastics and plasticizers.
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