Hepatic GCGR is required for the superior weight loss effects of a structurally related analogue of the dual GCGR/GLP1R agonist survodutide

Fen Long, Manuel Klug, Tenagne D. Challa, Vissarion Efthymiou, Christian Wolfrum, Carla Horvath
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Abstract

The dual glucagon/glucagon-like peptide 1 receptor (GCGR/GLP1R) agonists have superior efficacy in promoting weight loss and metabolic improvements in obesity and metabolic dysfunction-associated steatohepatitis (MASH) than current available mono-agonists. However, the mechanisms underlying these benefits are not fully understood. While the effects on appetite regulation and glucose control through GLP1R agonism are well established, the role of GCGR agonism in promoting weight loss and metabolic changes is less defined. Using a dual GCGR/GLP1R agonist BI 456908 and a selective GLP1R agonist semaglutide, we could show that the dual agonist achieved superior weight loss efficacy by engaging hepatic GCGR without adversely affecting glucose control. Furthermore, we could demonstrate that hepatic GCGR is critical for facilitating plasma and liver lipid clearance stimulated by the dual agonist. Overall, these findings highlight the crucial metabolic contributions of hepatic GCGR to the efficacy of combined GCGR/GL1R activation.
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GCGR/GLP1R 双重激动剂的结构相关类似物生存多肽具有卓越的减肥效果需要肝脏 GCGR 的参与
胰高血糖素/胰高血糖素样肽 1 受体(GCGR/GLP1R)双重激动剂在促进肥胖症和代谢功能障碍相关性脂肪性肝炎(MASH)患者减轻体重和改善代谢方面的疗效优于现有的单一激动剂。然而,这些益处的机制尚未完全明了。虽然 GLP1R 激动剂对食欲调节和血糖控制的作用已得到公认,但 GCGR 激动剂在促进减肥和新陈代谢变化方面的作用还不太明确。通过使用 GCGR/GLP1R 双激动剂 BI 456908 和选择性 GLP1R 激动剂 semaglutide,我们可以证明这种双激动剂通过参与肝脏 GCGR 而实现了卓越的减肥效果,同时不会对血糖控制产生不利影响。此外,我们还证明肝脏 GCGR 对于促进双重激动剂刺激下的血浆和肝脏脂质清除至关重要。总之,这些发现强调了肝脏 GCGR 对 GCGR/GL1R 联合激活疗效的重要代谢贡献。
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