Severe Hypoxemia Due to Elongation and Shunting of Patent Foramen Ovale After Emergent Repair of Thoracic Aortic Dissection

Gael Charbonne MD , Nicholas Whitmore DO , Damian Valencia MD , Raja Amir Nazir MD , Nathaniel Dittoe MD
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Abstract

We document the elongation and shunting of a patent foramen ovale (PFO) after thoracic aortic dissection repair in a 63-year-old man. Initially, a presurgical echocardiogram showed insignificant PFO shunting; however, severe hypoxemia and inability to extubate after thoracic aortic dissection repair necessitated further investigation. A repeat transesophageal echocardiogram after cardiothoracic surgery revealed significant PFO elongation with bidirectional shunting. Subsequent urgent transcatheter PFO closure markedly improved oxygenation, allowing for successful weaning from mechanical ventilation. This case highlights the importance of recognizing dynamic PFO changes after thoracic surgery as a reversible cause of postoperative hypoxemia.

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胸主动脉夹层急诊修补术后裂孔延长和分流导致的严重低氧血症
我们记录了一名 63 岁男子在胸主动脉夹层修补术后出现的卵圆孔未闭 (PFO) 延伸和分流。最初,手术前的超声心动图显示 PFO 分流不明显;然而,胸主动脉夹层修复术后出现严重低氧血症且无法拔管,因此有必要进行进一步检查。心胸手术后再次进行的经食道超声心动图检查显示,该患者的PFO明显延长,并伴有双向分流。随后紧急进行的经导管 PFO 关闭术明显改善了氧合,使患者成功脱离了机械通气。本病例强调了认识胸外科手术后 PFO 动态变化作为术后低氧血症可逆原因的重要性。
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CiteScore
1.40
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审稿时长
48 days
期刊最新文献
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