Age-related loss of intestinal barrier integrity plays an integral role in thymic involution and T cell ageing.

IF 8 1区 医学 Q1 CELL BIOLOGY Aging Cell Pub Date : 2024-11-15 DOI:10.1111/acel.14401
Jessica Conway, Erica N De Jong, Andrea J White, Ben Dugan, Nia Paddison Rees, Sonia M Parnell, Lisa E Lamberte, Archana Sharma-Oates, Jack Sullivan, Claudio Mauro, Willem van Schaik, Graham Anderson, Dawn M E Bowdish, Niharika A Duggal
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Abstract

The intestinal epithelium serves as a physical and functional barrier against harmful substances, preventing their entry into the circulation and subsequent induction of a systemic immune response. Gut barrier dysfunction has recently emerged as a feature of ageing linked to declining health, and increased intestinal membrane permeability has been shown to promote heightened systemic inflammation in aged hosts. Concurrent with age-related changes in the gut microbiome, the thymic microenvironment undergoes a series of morphological, phenotypical and architectural alterations with age, including disorganisation of the corticomedullary junction, increased fibrosis, increased thymic adiposity and the accumulation of senescent cells. However, a direct link between gut barrier dysbiosis and thymic involution leading to features of immune ageing has not been explored thus far. Herein, we reveal strong associations between enhanced microbial translocation and the peripheral accumulation of terminally differentiated, senescent and exhausted T cells and the compensatory expansion of regulatory T cells in older adults. Crucially, we demonstrate that aged germ-free mice are protected from age-related increases in intestinal permeability, highlighting the direct impact of mucosal permeability on thymic ageing. Together, these findings establish a novel mechanism by which gut barrier dysfunction drives systemic activation of the immune system during ageing through thymic involution. This enhances our understanding of drivers of T cell ageing and opens up the possibility for the use of microbiome-based interventions to restore immune homeostasis and promote healthy ageing in older adults.

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与年龄有关的肠道屏障完整性丧失在胸腺萎缩和 T 细胞老化中起着不可或缺的作用。
肠道上皮细胞是抵御有害物质的物理和功能屏障,防止有害物质进入血液循环,进而诱发全身免疫反应。肠道屏障功能失调是最近出现的一种与健康状况下降有关的老龄化特征,肠道膜通透性的增加已被证明会促进老龄宿主全身炎症的加剧。在肠道微生物组发生与年龄有关的变化的同时,胸腺微环境也会随着年龄的增长而发生一系列形态、表型和结构上的改变,包括皮质髓质交界处的紊乱、纤维化的增加、胸腺脂肪的增加和衰老细胞的堆积。然而,肠道屏障菌群失调与胸腺内陷导致免疫老化特征之间的直接联系迄今尚未得到探讨。在这里,我们揭示了微生物转运增强与终末分化、衰老和衰竭 T 细胞的外周积累以及老年人调节性 T 细胞的代偿性扩增之间的密切联系。最重要的是,我们证明了老年无菌小鼠可免受与年龄相关的肠道通透性增加的影响,这凸显了粘膜通透性对胸腺衰老的直接影响。这些发现共同建立了一种新的机制,即在衰老过程中,肠道屏障功能障碍通过胸腺内陷驱动免疫系统的系统激活。这加深了我们对 T 细胞老化驱动因素的理解,并为使用基于微生物的干预措施来恢复免疫平衡和促进老年人健康老化提供了可能性。
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来源期刊
Aging Cell
Aging Cell Biochemistry, Genetics and Molecular Biology-Cell Biology
自引率
2.60%
发文量
212
期刊介绍: Aging Cell is an Open Access journal that focuses on the core aspects of the biology of aging, encompassing the entire spectrum of geroscience. The journal's content is dedicated to publishing research that uncovers the mechanisms behind the aging process and explores the connections between aging and various age-related diseases. This journal aims to provide a comprehensive understanding of the biological underpinnings of aging and its implications for human health. The journal is widely recognized and its content is abstracted and indexed by numerous databases and services, which facilitates its accessibility and impact in the scientific community. These include: Academic Search (EBSCO Publishing) Academic Search Alumni Edition (EBSCO Publishing) Academic Search Premier (EBSCO Publishing) Biological Science Database (ProQuest) CAS: Chemical Abstracts Service (ACS) Embase (Elsevier) InfoTrac (GALE Cengage) Ingenta Select ISI Alerting Services Journal Citation Reports/Science Edition (Clarivate Analytics) MEDLINE/PubMed (NLM) Natural Science Collection (ProQuest) PubMed Dietary Supplement Subset (NLM) Science Citation Index Expanded (Clarivate Analytics) SciTech Premium Collection (ProQuest) Web of Science (Clarivate Analytics) Being indexed in these databases ensures that the research published in Aging Cell is discoverable by researchers, clinicians, and other professionals interested in the field of aging and its associated health issues. This broad coverage helps to disseminate the journal's findings and contributes to the advancement of knowledge in geroscience.
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