Correction: The LKB1-TSSK1B axis controls YAP phosphorylation to regulate the Hippo-YAP pathway.

IF 8.1 1区 生物学 Q1 CELL BIOLOGY Cell Death & Disease Pub Date : 2024-11-26 DOI:10.1038/s41419-024-07238-9
Cho-Long Kim, Su-Bin Lim, Sue-Hee Choi, Dong Hyun Kim, Ye Eun Sim, Eun-Hye Jo, Keeeun Kim, Keesook Lee, Hee-Sae Park, Su Bin Lim, Li-Jung Kang, Han-Sol Jeong, Youngsoo Lee, Carsten G Hansen, Jung-Soon Mo
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更正:LKB1-TSSK1B 轴控制 YAP 磷酸化,从而调节 Hippo-YAP 通路。
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来源期刊
Cell Death & Disease
Cell Death & Disease CELL BIOLOGY-
CiteScore
15.10
自引率
2.20%
发文量
935
审稿时长
2 months
期刊介绍: Brought to readers by the editorial team of Cell Death & Differentiation, Cell Death & Disease is an online peer-reviewed journal specializing in translational cell death research. It covers a wide range of topics in experimental and internal medicine, including cancer, immunity, neuroscience, and now cancer metabolism. Cell Death & Disease seeks to encompass the breadth of translational implications of cell death, and topics of particular concentration will include, but are not limited to, the following: Experimental medicine Cancer Immunity Internal medicine Neuroscience Cancer metabolism
期刊最新文献
Emerging mechanisms of lipid peroxidation in regulated cell death and its physiological implications. Microglial cannabinoid receptor type II stimulation improves cognitive impairment and neuroinflammation in Alzheimer's disease mice by controlling astrocyte activation. Correction: The LKB1-TSSK1B axis controls YAP phosphorylation to regulate the Hippo-YAP pathway. Inhibition of lung tumorigenesis by transient reprogramming in cancer cells. Enhancing mitochondrial one-carbon metabolism is neuroprotective in Alzheimer's disease models.
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