Erratum for the research article “The suppressive efficacy of THZ1 depends on KRAS mutation subtype and is associated with super-enhancer activity and the PI3K/AKT/mTOR signalling in pancreatic ductal adenocarcinoma: A hypothesis-generating study” by Huang et al

IF 7.9 1区 医学 Q1 MEDICINE, RESEARCH & EXPERIMENTAL Clinical and Translational Medicine Pub Date : 2024-12-27 DOI:10.1002/ctm2.70158
Lei Huang, Hui Yang, Kaidi Chen, Jing Yuan, Jie Li, Guanghai Dai, Mancang Gu, Yan Shi
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引用次数: 0

Abstract

Some inadvertent mistake in our paper entitled “The suppressive efficacy of THZ1 depends on KRAS mutation subtype and is associated with super-enhancer activity and the PI3K/AKT/mTOR signalling in pancreatic ductal adenocarcinoma: A hypothesis-generating study” [1] and published in Clinical and translational medicine came to our attention recently:

(1) There was an unintentional mistake in the placement and assembly of representative individual bioluminescence images in the original Figure 2A, which does not impact the statistical analysis, the described findings, or the conclusions. We would like to correct the representative individual bioluminescence image in the red block of the original Figure 2A as (Figure 1):

(2) There was an unintentional mistake in loading control band placement in Western blot, which does not influence the described findings or the conclusions. We would like to correct the loading control GAPDH in the red block of the original Figure 1I as (Figure 2).

And correct the loading control GAPDH in the red block of the original Figure 5C and E as (Figure 3):

The corrections do not have any impact on the described findings, the conclusions, or any other part of the manuscript. We sincerely apologize for any inconvenience caused.

Abstract Image

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Huang等人的研究文章《THZ1的抑制作用取决于KRAS突变亚型,并与胰腺导管腺癌中的超增强子活性和PI3K/AKT/mTOR信号传导相关:一项假设生成研究》的更正。
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来源期刊
CiteScore
15.90
自引率
1.90%
发文量
450
审稿时长
4 weeks
期刊介绍: Clinical and Translational Medicine (CTM) is an international, peer-reviewed, open-access journal dedicated to accelerating the translation of preclinical research into clinical applications and fostering communication between basic and clinical scientists. It highlights the clinical potential and application of various fields including biotechnologies, biomaterials, bioengineering, biomarkers, molecular medicine, omics science, bioinformatics, immunology, molecular imaging, drug discovery, regulation, and health policy. With a focus on the bench-to-bedside approach, CTM prioritizes studies and clinical observations that generate hypotheses relevant to patients and diseases, guiding investigations in cellular and molecular medicine. The journal encourages submissions from clinicians, researchers, policymakers, and industry professionals.
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