Systems neuroimmunology: current bottlenecks, research priorities and future directions

IF 27.6 1区 医学 Q1 IMMUNOLOGY Nature Immunology Pub Date : 2025-02-12 DOI:10.1038/s41590-025-02092-z
Harini Iyer, Christophe Benoist, Staci D. Bilbo, Lisa M. Boulanger, Michael D. Burton, Brian P. Daniels, Aleksandra Deczkowska, Martin F. Flajnik, Mélanie G. Gareau, Peter M. Grace, Javier E. Irazoqui, Susanna Rosi, Irene Salinas, Anne Schaefer, Caroline L. Sokol, Dionna W. Williams, Robyn S. Klein
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Abstract

Strategies to advance the field of neuroimmunology by embracing its complexity via inclusion of its multidisciplinary properties were discussed at a meeting in Cold Spring Harbor. Attendees proposed fostering of open communications and funding of collaborations across disciplines, and the recognition that our understanding of the neuroimmune system requires interdisciplinary science.

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系统神经免疫学:当前的瓶颈,研究重点和未来的方向
在冷泉港举行的一次会议上,讨论了通过包含其多学科特性来接受其复杂性以推进神经免疫学领域的策略。与会者建议促进开放交流,资助跨学科合作,并认识到我们对神经免疫系统的理解需要跨学科的科学。
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来源期刊
Nature Immunology
Nature Immunology 医学-免疫学
CiteScore
40.00
自引率
2.30%
发文量
248
审稿时长
4-8 weeks
期刊介绍: Nature Immunology is a monthly journal that publishes the highest quality research in all areas of immunology. The editorial decisions are made by a team of full-time professional editors. The journal prioritizes work that provides translational and/or fundamental insight into the workings of the immune system. It covers a wide range of topics including innate immunity and inflammation, development, immune receptors, signaling and apoptosis, antigen presentation, gene regulation and recombination, cellular and systemic immunity, vaccines, immune tolerance, autoimmunity, tumor immunology, and microbial immunopathology. In addition to publishing significant original research, Nature Immunology also includes comments, News and Views, research highlights, matters arising from readers, and reviews of the literature. The journal serves as a major conduit of top-quality information for the immunology community.
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Author Correction: Cleavage of roquin and regnase-1 by the paracaspase MALT1 releases their cooperatively repressed targets to promote TH17 differentiation. Tissue-specific clonal selection and differentiation of CD4⁺ T cells during infection. HLA class I signal peptide variation predicts strength of NKG2A+ NK cell response to missing-self and risk of human disease Lipid asymmetry disruption by XKR8 orchestrates neutrophil extracellular trap formation and inhibits fungal infection An inhibitory RNA checkpoint in TLR7 and TLR8.
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