{"title":"Work-Related Asthma Mortality, Michigan 2003-2023.","authors":"Kenneth D Rosenman, Mary Jo Reilly","doi":"10.1097/JOM.0000000000003377","DOIUrl":null,"url":null,"abstract":"<p><strong>Objective: </strong>To identify causes and factors associated with work-related asthma.</p><p><strong>Methods: </strong>There were 13 work-related asthma (WRA) deaths identified over 21 years in state-wide lung disease surveillance system.</p><p><strong>Results: </strong>The deceased ranged from 19 to 77. Eight had new-onset, and five had aggravated WRA. Five deaths from exposure to isocyanates, two to welding fumes, two to food products, and one death each from exposure to secondhand cigarette smoke, milk tank cleaning agents, chemicals used in construction, and molding machine release spray.</p><p><strong>Conclusions: </strong>Even when health care practioners note that work is a trigger of a patient's asthma, there is typically a delay in the recognition and action to address the workplace exposure(s). A WRA death is the ultimate consequence of a practioner's delay in not addressing work exposure(s) as an asthma trigger.</p>","PeriodicalId":94100,"journal":{"name":"Journal of occupational and environmental medicine","volume":" ","pages":""},"PeriodicalIF":0.0000,"publicationDate":"2025-02-28","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of occupational and environmental medicine","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.1097/JOM.0000000000003377","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
引用次数: 0
Abstract
Objective: To identify causes and factors associated with work-related asthma.
Methods: There were 13 work-related asthma (WRA) deaths identified over 21 years in state-wide lung disease surveillance system.
Results: The deceased ranged from 19 to 77. Eight had new-onset, and five had aggravated WRA. Five deaths from exposure to isocyanates, two to welding fumes, two to food products, and one death each from exposure to secondhand cigarette smoke, milk tank cleaning agents, chemicals used in construction, and molding machine release spray.
Conclusions: Even when health care practioners note that work is a trigger of a patient's asthma, there is typically a delay in the recognition and action to address the workplace exposure(s). A WRA death is the ultimate consequence of a practioner's delay in not addressing work exposure(s) as an asthma trigger.