RETRACTION: lncRNA NEAT1/miR-495-3p Regulates Angiogenesis in Burn Sepsis Through the TGF-β1 and SMAD Signaling Pathways

IF 2.7 4区 医学 Q3 IMMUNOLOGY Immunity, Inflammation and Disease Pub Date : 2025-03-21 DOI:10.1002/iid3.70177
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引用次数: 0

Abstract

RETRACTION: Y. Meng, Z. Hao, H. Zhang, P. Bai, W. Guo, X. Tian and J. Xu, “lncRNA NEAT1/miR-495-3p Regulates Angiogenesis in Burn Sepsis Through the TGF-β1 and SMAD Signaling Pathways,” Immunity, Inflammation and Disease 11, no. 1 (2023): e758, https://doi.org/10.1002/iid3.758.

The above article, published online on January 16, 2023 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement the journal Editor-in-Chief, Marc Veldhoen; and John Wiley & Sons Ltd. The retraction has been agreed due scientific flaws and inconsistencies found in the methodology and results of this article. While the authors were able to provide some supporting data, this was not sufficient and the irregularities remain. The editors have lost confidence in the results and conclusions presented in this study. The authors disagree with the retraction.

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结论:lncRNA NEAT1/miR-495-3p通过TGF-β1和SMAD信号通路调控烧伤脓毒症的血管生成
引用本文:张辉,郝振华,郭伟,徐军,郭伟,徐军,“通过TGF-β1和SMAD信号通路调控烧伤脓毒症血管生成的lncRNA NEAT1/miR-495-3p”,《免疫与炎症杂志》,第11期。1 (2023): e758, https://doi.org/10.1002/iid3.758。上述文章于2023年1月16日在线发表在Wiley在线图书馆(wileyonlinelibrary.com)上,经该杂志主编Marc Veldhoen同意撤回。及约翰威利父子有限公司。由于在本文的方法和结果中发现的科学缺陷和不一致,已同意撤回。虽然作者能够提供一些支持数据,但这是不够的,不正常现象仍然存在。编辑对本研究的结果和结论失去了信心。作者不同意撤稿。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
Immunity, Inflammation and Disease
Immunity, Inflammation and Disease Medicine-Immunology and Allergy
CiteScore
3.60
自引率
0.00%
发文量
146
审稿时长
8 weeks
期刊介绍: Immunity, Inflammation and Disease is a peer-reviewed, open access, interdisciplinary journal providing rapid publication of research across the broad field of immunology. Immunity, Inflammation and Disease gives rapid consideration to papers in all areas of clinical and basic research. The journal is indexed in Medline and the Science Citation Index Expanded (part of Web of Science), among others. It welcomes original work that enhances the understanding of immunology in areas including: • cellular and molecular immunology • clinical immunology • allergy • immunochemistry • immunogenetics • immune signalling • immune development • imaging • mathematical modelling • autoimmunity • transplantation immunology • cancer immunology
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