On a unified theory of cancer etiology and treatment based on the superconduction double-dipole model.

Abstract

The human biological cell is a complex nonlinear system that behaves electrically as a double dipole. The nonlinear property of the cytoplasmic membrane permits is to divide; but it is the double-dipole property that motivates division and growth. Increasing the double-dipole moment increases speed of division. If the time required for division due to forces developed by the double-dipole becomes much less than the time needed for the chromatin material of the nucleus to properly develop and mature, defective genes will be formed, producing mutated daughter cells. Thus any stimulus that for prolonged periods increases the double-dipole moment can be responsible for producing mutated cells. One such stimulus is a "supercurrent" from an organic superconducting source. This supercurrent applied to tissue increases the cellular dipole moment, hence can produce an uncontrolled proliferation of biological cells giving rise to a tumor. In contrast, an injury current produces a controlled proliferation of embryonic cells in the traumatized area of the system. The latter proliferation is regulated by the negative feedback action of the host, which does not occur in the case of the supercurrent produced by an organic superconductor. Knowledge of the kind of organic superconductor involved, its transition temperature, and the critical magnetic field could make feasible a therapy aimed at terminating the offending supercurrent in the host.