Implant design may influence delayed heterotopic ossification after total disk arthroplasty in lumbar spine

Eubulus J. Kerr MD, Ajay Jawahar MD, MS, Stephen Kay PA-C, David A. Cavanaugh MD, Pierce D. Nunley MD
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引用次数: 11

Abstract

Background

As total disk arthroplasty (TDA) gains increasing acceptance as an alternative to fusion for degenerative disk disease of the lumbar spine, new complications are encountered by the physicians during and after the procedure. We hereby report a complication after TDA in the lumbar spine that is in variance from previously proposed theories and suggests the possibility of implant design as one of the etiologic factors. The purpose of the present submission is to report a case of delayed heterotopic ossification (HO) after TDA that suggests that the keel-based design of the implant might have contributed to the etiology.

Case description

The patient underwent TDA for L3-4 degenerative disk disease and had fusion surgery for L5-S1 disease about 6 months later. During follow-up, development of significant HO was noticed at the L3 and L4 level. Radiologic studies revealed the origin of HO to be the keel cut made in the body of L3 to accommodate the keel-based artificial disk.

Conclusion

The exact etiology of HO after TDA is not clear. The presented anecdote points toward vertebral body trauma due to the design of the implant as a possible factor that needs to be studied more elaborately.

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植入物设计可能影响腰椎全椎间盘置换术后延迟异位骨化
背景:随着全椎间盘置换术(TDA)作为腰椎退变性椎间盘疾病融合的替代方法越来越被接受,医生在手术期间和手术后遇到了新的并发症。我们在此报告腰椎TDA术后的并发症,这与先前提出的理论不同,并提示植入物设计可能是病因之一。本报告的目的是报告一例TDA后延迟异位骨化(HO)的病例,这表明基于龙骨的植入物设计可能导致了病因。病例描述:患者因L3-4退变性椎间盘病变行TDA治疗,6个月后因L5-S1病变行融合手术。随访期间,在L3和L4水平发现了明显的HO。放射学研究显示,HO的起源是在L3体内的龙骨切割,以容纳基于龙骨的人工椎间盘。结论TDA后HO的确切病因尚不清楚。所提出的轶事表明,由于植入物的设计导致的椎体创伤可能是一个需要更详细研究的因素。
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Surgical Neurology
Surgical Neurology 医学-临床神经学
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