Exacerbation of Secondary Pulmonary Hypertension by Flat Chest after Lung Transplantation.

IF 1.1 4区 医学 Q4 CARDIAC & CARDIOVASCULAR SYSTEMS Annals of Thoracic and Cardiovascular Surgery Pub Date : 2022-08-20 Epub Date: 2020-11-03 DOI:10.5761/atcs.cr.20-00230
Haruhiko Shiiya, Masaaki Sato, Aya Shinozaki-Ushiku, Chihiro Konoeda, Kentaro Kitano, Jun Nakajima
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引用次数: 3

Abstract

A 40-year-old woman with idiopathic pleuroparenchymal fibroelastosis (IPPFE) and flat chest underwent left single lung transplantation (SLT). Although she had developed over-systemic pulmonary arterial pressure (PAP) at transplantation, it was alleviated. However, her PAP gradually increased again. Her transplanted lung was well-inflated, but progression of fibrosis in her right native lung appeared to have caused a mediastinal shift, and her flat chest caused obstruction of the outflow tract of the pulmonary vein. She died of heart failure and associated infection 1.5 years after transplantation. An autopsy confirmed irreversible pulmonary arterial and venous changes in the transplanted lung, suggestive of chronic pressure overload. The flat chest associated with IPPFE can affect pulmonary circulation after SLT.

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肺移植术后平胸加重继发性肺动脉高压的研究。
一位患有特发性胸膜实质纤维弹性增生症(IPPFE)和胸部扁平的40岁女性接受了左单肺移植(SLT)。虽然她在移植时出现过全身肺动脉压(PAP),但已减轻。然而,她的PAP又逐渐增加。她的移植肺充盈良好,但右肺纤维化的进展似乎导致纵隔移位,她平坦的胸部导致肺静脉流出道阻塞。她在移植后1.5年死于心力衰竭和相关感染。尸检证实移植肺不可逆的肺动脉和静脉改变,提示慢性压力超载。与IPPFE相关的平胸可影响SLT后的肺循环。
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来源期刊
Annals of Thoracic and Cardiovascular Surgery
Annals of Thoracic and Cardiovascular Surgery CARDIAC & CARDIOVASCULAR SYSTEMS-SURGERY
CiteScore
2.80
自引率
0.00%
发文量
56
审稿时长
4-8 weeks
期刊介绍: Information not localized
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