Sunil Gurmukhani, Preeti Gahlan, Sanjay Shah, Tejas M. Patel
{"title":"Isolated Unilateral Renal Artery Stenosis in Young Female with Takayasu Arteritis: Case Report","authors":"Sunil Gurmukhani, Preeti Gahlan, Sanjay Shah, Tejas M. Patel","doi":"10.15713/ins.johtn.0166","DOIUrl":null,"url":null,"abstract":"3 rd day of hospitalization� In view of young hypertension and recurrent flash pulmonary edema, we suspected renovascular etiology. On blood investigation, her Hb was 12.3, total count 12,300, platelets 3�2 lac, serum creatinine 0�7, serum potassium 4�2, and serum sodium 142� She had high plasma renin activity (>24) and high aldosterone level� Erythrocyte sedimentation rate (ESR) was 112 and C-reactive protein (CRP) was 98� Ultrasono graphy abdomen showed asymmetrical kidney size� Computerized tomograph y angiogram revealed diffuse thickening and enhancement of aortic wall and its major branches along with critical narrowing of ostium of left renal artery [Figure Atherosclerotic RAS is extremely unknown at this age� We put two differential diagnosis, one is unifocal FMD and other is Abstract Vasculitis as a cause of renovascular hypertension is not uncommon. However, isolated involvement of the left renal artery without affection of other vascular beds is extremely rare in any vasculitis, including Takayasu arteritis. Here, we present a case of a young girl with resistant hypertension and recurrent flash pulmonary edema secondary to renal artery stenosis (RAS). The cause of the RAS was vasculitis probably Takayasu arteritis. She was managed with immunosuppression with endovascular intervention.","PeriodicalId":38918,"journal":{"name":"Open Hypertension Journal","volume":"17 1","pages":""},"PeriodicalIF":0.0000,"publicationDate":"2019-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Open Hypertension Journal","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.15713/ins.johtn.0166","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q4","JCRName":"Medicine","Score":null,"Total":0}
引用次数: 0
Abstract
3 rd day of hospitalization� In view of young hypertension and recurrent flash pulmonary edema, we suspected renovascular etiology. On blood investigation, her Hb was 12.3, total count 12,300, platelets 3�2 lac, serum creatinine 0�7, serum potassium 4�2, and serum sodium 142� She had high plasma renin activity (>24) and high aldosterone level� Erythrocyte sedimentation rate (ESR) was 112 and C-reactive protein (CRP) was 98� Ultrasono graphy abdomen showed asymmetrical kidney size� Computerized tomograph y angiogram revealed diffuse thickening and enhancement of aortic wall and its major branches along with critical narrowing of ostium of left renal artery [Figure Atherosclerotic RAS is extremely unknown at this age� We put two differential diagnosis, one is unifocal FMD and other is Abstract Vasculitis as a cause of renovascular hypertension is not uncommon. However, isolated involvement of the left renal artery without affection of other vascular beds is extremely rare in any vasculitis, including Takayasu arteritis. Here, we present a case of a young girl with resistant hypertension and recurrent flash pulmonary edema secondary to renal artery stenosis (RAS). The cause of the RAS was vasculitis probably Takayasu arteritis. She was managed with immunosuppression with endovascular intervention.