{"title":"Acute hepatic failure with hemolytic anemia due to Hepatitis A infection with coexistent glucose-6-phosphate dehydrogenase deficiency","authors":"Akash Bhutra , Prabhas Prasun Giri , Nupur Ganguly","doi":"10.1016/j.pid.2015.04.002","DOIUrl":null,"url":null,"abstract":"<div><p><span>Though only 0.1% of acute viral Hepatitis<span><span> A complicated as acute hepatic failure, Hepatitis A has been detected as the most common aetiology of acute hepatic failure in India. Any co-existence morbidity like </span>G6PD deficiency<span> certainly makes it more complicated. Here we present a case where a 10 year old boy admitted with Acute Hepatic Failure with grade 3 encephalopathy with severe hyperbilirubinemia<span> and haemolytic anemia with features of </span></span></span></span>acute tubular necrosis and later on found to be G6PD deficient.</p></div>","PeriodicalId":19984,"journal":{"name":"Pediatric Infectious Disease","volume":"7 2","pages":"Pages 50-52"},"PeriodicalIF":0.0000,"publicationDate":"2015-04-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1016/j.pid.2015.04.002","citationCount":"2","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Pediatric Infectious Disease","FirstCategoryId":"1085","ListUrlMain":"https://www.sciencedirect.com/science/article/pii/S2212832815000314","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
引用次数: 2
Abstract
Though only 0.1% of acute viral Hepatitis A complicated as acute hepatic failure, Hepatitis A has been detected as the most common aetiology of acute hepatic failure in India. Any co-existence morbidity like G6PD deficiency certainly makes it more complicated. Here we present a case where a 10 year old boy admitted with Acute Hepatic Failure with grade 3 encephalopathy with severe hyperbilirubinemia and haemolytic anemia with features of acute tubular necrosis and later on found to be G6PD deficient.