The rôle of speciation in mercury methylation in sediments and water

Environmental Pollution Series B, Chemical and Physical Pub Date : 1985-01-01 DOI:10.1016/0143-148X(85)90011-4

P.J. Craig, P.A. Moreton

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引用次数: 35

Abstract

The rate and extent of the methylation of inorganic mercury has been shown to depend markedly on the chemical speciation of the mercury and the methylating agent. The presence of mercury (II)—chlorine covalent bonding has an inhibitory effect on methylation by methyl cobalamin (CH₃CoB₁₂) and in natural sediments. The rôle of radical and carbonium ion methylation mechanisms has also been investigated. In a typical estuarine sediment the extent of mercury methylation is as follows: Hg(OAc)₂ ⪢ Hg(cyst)₂ > Hg(cyst) (Hcyst)Cl > Hg(meth)₂²⁺ > Hg(eth)₂²⁺ > Hg(Hcyst)Cl₂ > HgCl₂ > (HgCl₂)₂ pen, where OAc = acetateHcyst = cysteine, meth = methionine, eth = ethionine and pen = penicillamine. This is consistent with mercury methylation arising by attack of a methyl carbanion (CH₃⁻) on inorganic mercury (II) ions derived by microbial breakdown of the original and mercury compounds added.

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沉积物和水中汞甲基化的物种形成rôle

无机汞甲基化的速度和程度已被证明明显取决于汞和甲基化剂的化学形态。汞(II) -氯共价键的存在对甲基钴胺素(CH3CoB12)和天然沉积物中的甲基化具有抑制作用。研究了rôle自由基甲基化和碳离子甲基化的机理。典型河口沉积物中汞甲基化程度如下:Hg(OAc)2⪢Hg(囊肿)2 >Hg(囊肿)(h囊肿)Cl >Hg(冰毒)22 +比;Hg (eth) 22 +比;Hg (Hcyst)氯比;HgCl2祝辞(HgCl2)2笔，其中OAc = acetatehcyth =半胱氨酸，meth =蛋氨酸，eth =蛋氨酸，pen =青霉胺。这与汞甲基化是一致的，甲基碳离子(CH3−)攻击无机汞(II)离子产生的汞甲基化是由微生物分解原始和添加的汞化合物。

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