钠耗尽犬肾性高血压发展过程中心排血量的变化。

J O Davis, G A Stephens, R H Freeman, J M Deforrest
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引用次数: 2

摘要

1. 慢性肾血管性高血压的发展在未切除肾,钠耗尽狗与减少心输出量。2. 随着钠和容量的补充,这些动物的心输出量恢复正常,但高水平的动脉压没有改变;结果外周小动脉扩张。3.这些观察结果为反对全身自动调节理论提供了证据。
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Changes in cardiac output during the development of renal hypertension in sodium-depleted dogs.

1. Chronic renovascular hypertension developed in uninephrectomized, sodium-depleted dogs in association with a decrease in cardiac output. 2. With sodium and volume repletion of these animals, cardiac output returned to normal but the high level of arterial pressure was unchanged; consequently, the peripheral arterioles dilated. 3. These observations provide evidence against the theory of whole-body autoregulation.

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Proceedings of the Fifth Meeting of the International Society of Hypertension, Paris, 12-14 June 1978. Brain catecholamines and catecholamine-synthesizing enzymes in renovascular hypertension in the rat. Enhanced hypothalamic noradrenaline biosynthesis in Goldblatt I renovascular hypertension. Definitive evidence for renin in rat brain by affinity chromatographic separation from protease. Renal release of active and inactive renin in essential and renovascular hypertension.
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