下肢压迫性神经病的外科治疗

R. Tiel
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引用次数: 4

摘要

周围神经在其行进过程中的任何一点都可能受损。当直接创伤是原因时,很少有意外,但有时神经功能障碍是自发发生的,没有明确的外部创伤的证据。在这种情况下,临床医生寻找其他解释,外科医生寻找解剖原因进行治疗。在没有外伤的情况下,卡压性神经病变通常被认为是鉴别诊断。通常认为,压迫性神经病是一种外部收缩过程,通过施加局灶压力,导致周围神经功能障碍。不幸的是,这种理解——尽管在概念上对最常见的局灶性神经病变,腕管综合征有用——在机械和几何上过于有限,不能正确地适用于其他局灶性神经病变。不幸的是,“诱捕”一词在医学术语中似乎根深蒂固,目前无法取代。局灶性神经病适合手术治疗,似乎提供了一个更好的清单,并不局限于涉及结缔组织的特定几何形状。这篇文章的重点是手术解决那些特定的和特征性的位置在下肢,解剖和它的变化相交以外的神经功能的生理界限,并允许局灶性神经损伤发生,并成为临床明显。一般来说,周围神经系统的神经受到很好的保护,不受外力的影响。神经在不造成结构损伤的情况下进行伸展的能力已经被人们认识了很多年,就像它承受压力的能力一样。随着认识到这一点,人们意识到不对称的施加力和压力梯度可能引起一系列的结构和生理变化,从而导致神经及其纤维丧失功能。这些变化有的具有宏观表现,如局灶性
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The Surgical Treatment of Entrapment Neuropathies of the Lower Extremity
A peripheral nerve may be damaged at any point along its course. When direct trauma is the cause there are few surprises, but on occasion nerve dysfunction occurs spontaneously without clear evidence of ex-ternal trauma. In these situations the clinician searches for other explanations and the surgeon looks for an anatomic cause to treat. In the absence of trauma, entrapment neuropathy is often considered in the differential diagnosis. As usually understood, entrapment neuropathy is an external constrictive process that, by the application of focal pressure, is responsible for the resultant dysfunction of a given peripheral nerve. Unfortunately, this understanding—although conceptually useful for the most common focal neuropathy, carpal tunnel syn-drome—is too limited mechanistically and geometrically to lend itself correctly to other focal mechanical neuropathies. Unfortunately, the term entrapment seems too well ingrained in medical parlance to be displaced at this time. Focal neuropathies amenable to surgical solutions would seem to offer a better list and are not limited to a particular geometry involving connective tissue. This article focuses on surgical solutions to those specific and characteristic locations in the lower extremity where anatomy and its variations intersect outside the physiologic boundaries of nerve function and allow focal nerve damage to occur and become clinically evident. It may be stated axomatically that the nerves of the peripheral nervous system are, in general, well protected from external forces. The ability of nerve to stretch somewhat without structural damage has been recognized for many years, 1,2 as has its ability to with-stand some pressure. 3 Along with this recognition has come the awareness that asymmetrically applied forces and pressure gradients may set in motion a series of structural and physiologic changes that will cause the nerve and its fibers to fail in function. Some of theses changes have macroscopic manifestations, such as focal
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