人冠状动脉成形术后再狭窄早期增殖血管平滑肌细胞氧化应激表达升高

T. Oigawa, Hiromasa Suzuki, S. Sunayama, Sachio KAWAIl, H. Daida
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引用次数: 0

摘要

血管损伤的修复是一个涉及复杂信号通路的协同反应,是血管成形术后再狭窄的临床特征之一。在再狭窄的过程中,血管损伤导致各种生物细胞的激活和许多血管活性、血栓形成和有丝分裂因子的释放。再狭窄在组织学上归因于新生内膜增生
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Increased Expression of Oxidative Stress in Proliferated Vascular Smooth Muscle Cells in Early Stage of Human Restenosis After Coronary Angioplasty
The repair of vascular injury is a concerted response involving complex signaling pathway that contributes to the clinical feature of post─angioplasty restenosis . Vascular injury leads to the activation of various biological cells and the release of numerous vasoactive, thrombogenic, and mitogenic factors in the process of restenosis. Restenosis is histologically attributed to neointimal hyperplasia Original Articles
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