5型心肌梗死1例

Y. Tunitsky-Lifshitz, A. Braun, G. Segal
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引用次数: 0

摘要

尽管在介入心脏病学和胸外科领域取得了重大技术进步,围手术期心肌梗死(MI)仍然很常见(1-5)。大多数关于过程中心肌梗死的文献都是十年前写的。试图定义冠状动脉旁路手术后临床相关的MI定义增加≥10 x在心脏生物标记作为切断,以防preprocedural肌钙蛋白是已知的,或正常肌钙蛋白≥70 x值(5)。在此,我们描述一个案件的报道说有一名妇女,来到我们医院的康复中心后冠状动脉旁路移植(CABG)是在另一个农村医院,与海拔≥60 x肌钙蛋白从急诊室的基线(ER)。对她进行评估的心脏病专家将其归因于残余心肌损伤,并让她住进了内科病房。仅在第二天再次进行肌钙蛋白检测后,诊断为5型心肌梗死,并进行了紧急冠状动脉造影,显示静脉移植物完全血栓形成。
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Type 5 myocardial infarction: A case report
Periprocedural myocardial infarction (MI) remain common despite major technical advances in the field of interventional cardiology and thoracic surgery (1-5). Most of the literature regarding periprocedural MI was written about a decade ago. Attempts to define clinically relevant MI after coronary bypass define a rise of ≥ 10x in cardiac biomarkers as a cutoff in case the preprocedural troponin is known, or ≥70x of normal troponin values (5). Herein, we describe a case report of a woman that came to a rehabilitation center in our hospital after a Coronary Artery Bypass Graft (CABG) was done in another rural hospital, with an elevation of ≥60 x troponin from baseline in the emergency room (ER). The cardiologist that assessed her attributed the rise to remnant myocardial damage and admitted her to an internal ward. Only after another troponin test taken the following day, the diagnosis of type 5 MI was made and an urgent coronary angiography was done, revealing a complete thrombosis of her venous graft.
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