Erdr1通过与YAP1和Mid1的动态相互作用驱动巨噬细胞编程

Q3 Medicine ImmunoHorizons Pub Date : 2024-02-01 DOI:10.4049/immunohorizons.2400004
Yuhang Wang
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引用次数: 0

摘要

红细胞分化调节因子 1(Erdr1)是一种应激诱导的、广泛表达的、高度保守的分泌因子,在人类和小鼠中均有发现。Erdr1 与 Hippo-YAP1 信号传导有关。Erdr1 最初被认为是血红蛋白合成的诱导因子,后来成为一种多功能蛋白质,尤其是在免疫细胞中。虽然 Erdr1 与 T 细胞和 NK 细胞功能的调节有关,但它在巨噬细胞中的作用仍不清楚。本研究探讨了 Erdr1 在巨噬细胞炎症反应中的功能和机制。数据显示,Erdr1能促进抗炎细胞因子的产生,这也是之前研究报道过的功能。然而,我发现 Erdr1 也能发挥促炎作用。Erdr1 在巨噬细胞中的功能取决于其剂量和细胞密度。我观察到,与未极化的巨噬细胞相比,Erdr1 在 M1 巨噬细胞中的表达受到抑制,但在 M2 巨噬细胞中则上调。我推测 Erdr1 通过与不同浓度的适配体结合来平衡炎症反应。从机理上讲,我证明了 YAP1 和 Mid1 是 Erdr1 的两种适配蛋白。当 Erdr1 水平升高时,Erdr1-YAP1 相互作用会促进抗炎细胞因子的产生;而当 Erdr1 水平降低时,Erdr1-Mid1 相互作用会诱导促炎细胞因子的产生。这项研究强调了 Erdr1 对极化巨噬细胞产生细胞因子的调节作用,可能是通过调节非经典 Hippo 通路中的 YAP1 来实现的。
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Erdr1 Drives Macrophage Programming via Dynamic Interplay with YAP1 and Mid1.

Erythroid differentiation regulator 1 (Erdr1) is a stress-induced, widely expressed, highly conserved secreted factor found in both humans and mice. Erdr1 is linked with the Hippo-YAP1 signaling. Initially identified as an inducer of hemoglobin synthesis, Erdr1 emerged as a multifunctional protein, especially in immune cells. Although Erdr1 has been implicated in regulating T cells and NK cell function, its role in macrophage remains unclear. This study explored the function and mechanism of Erdr1 in macrophage inflammatory response. The data demonstrated that Erdr1 could promote anti-inflammatory cytokine production, a function that also has been reported by previous research. However, I found Erdr1 also could play a proinflammatory role. The function of Erdr1 in macrophages depends on its dose and cell density. I observed that Erdr1 expression was inhibited in M1 macrophages but was upregulated in M2 macrophages compared with unpolarized macrophages. I hypothesized that Erdr1 balances the inflammatory response by binding with distinct adaptors dependent on varying concentrations. Mechanistically, I demonstrated YAP1 and Mid1 as the two adaptor proteins of Erdr1. The Erdr1-YAP1 interaction promotes anti-inflammatory cytokine production when Erdr1 levels are elevated, whereas the Erdr1-Mid1 interaction induces proinflammatory cytokine production when Erdr1 levels are decreased. This study highlights the effects of Erdr1 on regulating cytokine production from polarized macrophages potentially by regulating YAP1 in the nonclassical Hippo pathway.

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