钙蛋白酶-2抑制剂NA-184的鉴定和神经保护特性

IF 2.9 4区 医学 Q2 PHARMACOLOGY & PHARMACY Pharmacology Research & Perspectives Pub Date : 2024-04-01 DOI:10.1002/prp2.1181
Michel Baudry, Yubin Wang, Xiaoning Bi, Yun Lyna Luo, Zhijun Wang, Zeechan Kamal, Alexander Shirokov, Ed Sullivan, Dennis Lagasca, Hany Khalil, Gary Lee, Kathy Fosnaugh, Philippe Bey, Shujaath Mehdi, Greg Coulter
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引用次数: 0

摘要

我们的实验室已经证明,急性损伤后大脑中的钙蛋白酶-2活化与神经元损伤和损伤的长期功能后果直接相关,而钙蛋白酶-1活化通常具有神经保护作用,钙蛋白酶-1缺失会加剧神经元损伤。我们的研究还表明,一种相对选择性的钙蛋白酶-2抑制剂(称为C2I)可增强小鼠的长期电位、学习和记忆,并在受控皮层冲击(CCI)的创伤性脑损伤(TBI)模型中提供神经保护。利用分子动力学模拟和配体竞争饱和位点识别(SILCS)软件,我们生成了约 130 种 C2I 类似物,并在一系列体外和体内试验中对它们进行了测试。结果发现了两种有趣的化合物:NA-112 和 NA-184。进一步的分析表明,NA-184((S)-2-(3-苄脲基)-N-((R,S)-1-((3-氯-2-甲氧基苄基)氨基)-1,2-二氧代戊烷-3-基)-4-甲基戊酰胺)在小鼠脑组织和人类细胞系中的测试浓度下,可选择性地、剂量依赖性地抑制钙蛋白酶-2的活性,而对钙蛋白酶-1没有明显的抑制作用。与 NA-112 一样,NA-184 也能抑制 TBI 诱导的钙蛋白酶-2 激活和雌雄小鼠和大鼠的细胞死亡。药代动力学和药效学分析表明,NA-184表现出的特性,包括在血浆和肝脏中的稳定性以及血脑屏障渗透性,使其成为治疗创伤性脑损伤的临床候选药物。
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Identification and neuroprotective properties of NA-184, a calpain-2 inhibitor.

Our laboratory has shown that calpain-2 activation in the brain following acute injury is directly related to neuronal damage and the long-term functional consequences of the injury, while calpain-1 activation is generally neuroprotective and calpain-1 deletion exacerbates neuronal injury. We have also shown that a relatively selective calpain-2 inhibitor, referred to as C2I, enhanced long-term potentiation and learning and memory, and provided neuroprotection in the controlled cortical impact (CCI) model of traumatic brain injury (TBI) in mice. Using molecular dynamic simulation and Site Identification by Ligand Competitive Saturation (SILCS) software, we generated about 130 analogs of C2I and tested them in a number of in vitro and in vivo assays. These led to the identification of two interesting compounds, NA-112 and NA-184. Further analyses indicated that NA-184, (S)-2-(3-benzylureido)-N-((R,S)-1-((3-chloro-2-methoxybenzyl)amino)-1,2-dioxopentan-3-yl)-4-methylpentanamide, selectively and dose-dependent inhibited calpain-2 activity without evident inhibition of calpain-1 at the tested concentrations in mouse brain tissues and human cell lines. Like NA-112, NA-184 inhibited TBI-induced calpain-2 activation and cell death in mice and rats, both male and females. Pharmacokinetic and pharmacodynamic analyses indicated that NA-184 exhibited properties, including stability in plasma and liver and blood-brain barrier permeability, that make it a good clinical candidate for the treatment of TBI.

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来源期刊
Pharmacology Research & Perspectives
Pharmacology Research & Perspectives Pharmacology, Toxicology and Pharmaceutics-General Pharmacology, Toxicology and Pharmaceutics
CiteScore
5.30
自引率
3.80%
发文量
120
审稿时长
20 weeks
期刊介绍: PR&P is jointly published by the American Society for Pharmacology and Experimental Therapeutics (ASPET), the British Pharmacological Society (BPS), and Wiley. PR&P is a bi-monthly open access journal that publishes a range of article types, including: target validation (preclinical papers that show a hypothesis is incorrect or papers on drugs that have failed in early clinical development); drug discovery reviews (strategy, hypotheses, and data resulting in a successful therapeutic drug); frontiers in translational medicine (drug and target validation for an unmet therapeutic need); pharmacological hypotheses (reviews that are oriented to inform a novel hypothesis); and replication studies (work that refutes key findings [failed replication] and work that validates key findings). PR&P publishes papers submitted directly to the journal and those referred from the journals of ASPET and the BPS
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