{"title":"自噬通过AMP激活的蛋白激酶/哺乳动物雷帕霉素靶点通路调节缺氧条件下骨髓间充质干细胞的凋亡》的更正。","authors":"","doi":"10.1002/cbin.12142","DOIUrl":null,"url":null,"abstract":"<p>Zhang, Z., Yang, M., Wang, Y., Wang, L., Jin, Z., Ding, L., Zhang, L., Zhang, L., Jiang, W., Gao, G., Yang, J., Lu, B., Cao, F., & Hu, T. (2016) Autophagy regulates the apoptosis of bone marrow-derived mesenchymal stem cells under hypoxic condition via AMP-activated protein kinase/mammalian target of rapamycin pathway. <i>Cell Biology Internationa</i>, 40(6), 671–685. https://doi.org/10.1002/cbin.10604.</p><p>In the articles, the following figures are corrected as</p><p>I. Additional information/correction for Figure 5a. In the original version of our article, an oversight occurred during the assembly of figures, leading to the inadvertent inclusion of an incorrect image in the \"Hypoxia+, 3-MA-, and Rapamycin-\" group within Figure 5A. We extend our apologies for any misunderstanding this may have caused. We wish to clarify that this oversight does not impact the manuscript's quantitative analyses or conclusions. We have addressed this error and the corrected Figure 5A is presented below.</p><p>II. Additional information/correction for Figure 7f. In the first publication of our paper, there was an unintended mistake in the compilation of the figures, resulting in the wrong image being used in Figure 7F. We apologize for any confusion this error may have led to. We want to assure our readers that this oversight did not influence the quantitative analysis or the findings of our study. The corrected Figure 7F has been provided. We sincerely regret any disturbance or inconvenience this mistake might have caused.</p>","PeriodicalId":9806,"journal":{"name":"Cell Biology International","volume":"48 5","pages":"755-756"},"PeriodicalIF":3.1000,"publicationDate":"2024-03-11","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1002/cbin.12142","citationCount":"0","resultStr":"{\"title\":\"Correction to “Autophagy regulates the apoptosis of bone marrow-derived mesenchymal stem cells under hypoxic condition via AMP-activated protein kinase/mammalian target of rapamycin pathway”\",\"authors\":\"\",\"doi\":\"10.1002/cbin.12142\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p>Zhang, Z., Yang, M., Wang, Y., Wang, L., Jin, Z., Ding, L., Zhang, L., Zhang, L., Jiang, W., Gao, G., Yang, J., Lu, B., Cao, F., & Hu, T. (2016) Autophagy regulates the apoptosis of bone marrow-derived mesenchymal stem cells under hypoxic condition via AMP-activated protein kinase/mammalian target of rapamycin pathway. <i>Cell Biology Internationa</i>, 40(6), 671–685. https://doi.org/10.1002/cbin.10604.</p><p>In the articles, the following figures are corrected as</p><p>I. Additional information/correction for Figure 5a. In the original version of our article, an oversight occurred during the assembly of figures, leading to the inadvertent inclusion of an incorrect image in the \\\"Hypoxia+, 3-MA-, and Rapamycin-\\\" group within Figure 5A. We extend our apologies for any misunderstanding this may have caused. We wish to clarify that this oversight does not impact the manuscript's quantitative analyses or conclusions. We have addressed this error and the corrected Figure 5A is presented below.</p><p>II. Additional information/correction for Figure 7f. In the first publication of our paper, there was an unintended mistake in the compilation of the figures, resulting in the wrong image being used in Figure 7F. We apologize for any confusion this error may have led to. We want to assure our readers that this oversight did not influence the quantitative analysis or the findings of our study. The corrected Figure 7F has been provided. We sincerely regret any disturbance or inconvenience this mistake might have caused.</p>\",\"PeriodicalId\":9806,\"journal\":{\"name\":\"Cell Biology International\",\"volume\":\"48 5\",\"pages\":\"755-756\"},\"PeriodicalIF\":3.1000,\"publicationDate\":\"2024-03-11\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://onlinelibrary.wiley.com/doi/epdf/10.1002/cbin.12142\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Cell Biology International\",\"FirstCategoryId\":\"99\",\"ListUrlMain\":\"https://onlinelibrary.wiley.com/doi/10.1002/cbin.12142\",\"RegionNum\":3,\"RegionCategory\":\"生物学\",\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"Q3\",\"JCRName\":\"CELL BIOLOGY\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Cell Biology International","FirstCategoryId":"99","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1002/cbin.12142","RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q3","JCRName":"CELL BIOLOGY","Score":null,"Total":0}
引用次数: 0
摘要
Zhang,Z.,Yang,M.,Wang,Y.,Wang,L.,Jin,Z.,Ding,L.,Zhang,L.,Zhang,L.,Jiang,W.,Gao,G.,Yang,J.,Lu,B.,Cao,F、& Hu, T. (2016) Autophagy regulates the apoptosis of bone marrow-derived mesenchymal stem cells under hypoxic condition via AMP-activated protein kinase/mammalian target of rapamycin pathway.国际细胞生物学》(Cell Biology Internationa),40(6),671-685。https://doi.org/10.1002/cbin.10604.In,以下图表更正为I.图 5a 的补充信息/更正。在我们文章的原始版本中,由于组装图时的疏忽,导致图 5A 中的 "缺氧+、3-MA- 和雷帕霉素-"组无意中包含了一张错误的图片。对于由此可能造成的误解,我们深表歉意。我们希望澄清,这一疏忽并不影响手稿的定量分析或结论。我们已经纠正了这一错误,更正后的图 5A 见下文。图 7f 的补充信息/更正。在我们首次发表的论文中,由于在编译图表时出现意外错误,导致图 7F 中使用了错误的图片。我们对这一错误可能导致的任何混淆表示歉意。我们向读者保证,这一疏忽并没有影响我们的定量分析或研究结果。更正后的图 7F 已经提供。我们对这一错误可能造成的任何干扰或不便表示诚挚的歉意。
Correction to “Autophagy regulates the apoptosis of bone marrow-derived mesenchymal stem cells under hypoxic condition via AMP-activated protein kinase/mammalian target of rapamycin pathway”
Zhang, Z., Yang, M., Wang, Y., Wang, L., Jin, Z., Ding, L., Zhang, L., Zhang, L., Jiang, W., Gao, G., Yang, J., Lu, B., Cao, F., & Hu, T. (2016) Autophagy regulates the apoptosis of bone marrow-derived mesenchymal stem cells under hypoxic condition via AMP-activated protein kinase/mammalian target of rapamycin pathway. Cell Biology Internationa, 40(6), 671–685. https://doi.org/10.1002/cbin.10604.
In the articles, the following figures are corrected as
I. Additional information/correction for Figure 5a. In the original version of our article, an oversight occurred during the assembly of figures, leading to the inadvertent inclusion of an incorrect image in the "Hypoxia+, 3-MA-, and Rapamycin-" group within Figure 5A. We extend our apologies for any misunderstanding this may have caused. We wish to clarify that this oversight does not impact the manuscript's quantitative analyses or conclusions. We have addressed this error and the corrected Figure 5A is presented below.
II. Additional information/correction for Figure 7f. In the first publication of our paper, there was an unintended mistake in the compilation of the figures, resulting in the wrong image being used in Figure 7F. We apologize for any confusion this error may have led to. We want to assure our readers that this oversight did not influence the quantitative analysis or the findings of our study. The corrected Figure 7F has been provided. We sincerely regret any disturbance or inconvenience this mistake might have caused.
期刊介绍:
Each month, the journal publishes easy-to-assimilate, up-to-the minute reports of experimental findings by researchers using a wide range of the latest techniques. Promoting the aims of cell biologists worldwide, papers reporting on structure and function - especially where they relate to the physiology of the whole cell - are strongly encouraged. Molecular biology is welcome, as long as articles report findings that are seen in the wider context of cell biology. In covering all areas of the cell, the journal is both appealing and accessible to a broad audience. Authors whose papers do not appeal to cell biologists in general because their topic is too specialized (e.g. infectious microbes, protozoology) are recommended to send them to more relevant journals. Papers reporting whole animal studies or work more suited to a medical journal, e.g. histopathological studies or clinical immunology, are unlikely to be accepted, unless they are fully focused on some important cellular aspect.
These last remarks extend particularly to papers on cancer. Unless firmly based on some deeper cellular or molecular biological principle, papers that are highly specialized in this field, with limited appeal to cell biologists at large, should be directed towards journals devoted to cancer, there being very many from which to choose.