血管内时代慢性 B 型主动脉夹层开放式修复术的中长期效果。

IF 1.4 4区 医学 Q3 CARDIAC & CARDIOVASCULAR SYSTEMS Heart and Vessels Pub Date : 2024-09-01 Epub Date: 2024-03-27 DOI:10.1007/s00380-024-02399-1
Akitoshi Takazawa, Toshihisa Asakura, Osamu Kinoshita, Hiroyuki Nakajima, Akihiro Yoshitake
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引用次数: 0

摘要

药物治疗是慢性 B 型主动脉夹层(CTBAD)的标准治疗方法。然而,开胸手术修复(OSR)和胸腔内血管修复(TEVAR)在 CTBAD 患者中的作用仍存在争议。因此,本研究旨在评估和比较经左侧开胸手术进行 OSR 与 TEVAR 治疗 CTBAD 的中长期临床疗效。研究回顾性分析了 2007 年 4 月至 2021 年 5 月期间连续接受 CTBAD 手术治疗的 85 例患者的数据。患者被分为两组:G组包括接受OSR手术的患者,E组包括接受TEVAR手术的患者。G 组和 E 组分别有 33 名和 52 名患者。对术前和术后计算机断层扫描(CT)检查的最大直径进行了回顾性分析。平均随访时间为 5.8 年。没有出现手术死亡率。中风(G:2 对 E:0,P = 0.30)、截瘫(G:1 对 E:1,P = 0.66)和呼吸衰竭(G:2 对 E:0,P = 0.30)等并发症没有差异。观察到的术前因素差异包括发病与手术之间的间隔时间(G;4.9 年 vs. E;1.9 年,p
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Mid- and long-term results of open repair for chronic type B aortic dissection in endovascular era.

Medical management is the standard treatment of chronic type B aortic dissection (CTBAD). However, the roles of open surgical repair (OSR) and thoracic endovascular repair (TEVAR) in patients with CTBAD remain controversial. Thus, this study aimed to assess and compare the mid- and long-term clinical outcomes of OSR via left thoracotomy with that of TEVAR for CTBAD. The data of 85 consecutive patients who underwent surgery for CTBAD from April 2007 to May 2021 were retrospectively reviewed. The patients were divided into two groups: Group G, which included patients who underwent OSR, and Group E, which included patients who underwent TEVAR. Groups G and E comprised 33 and 52 patients, respectively. Preoperative and postoperative computed tomography (CT) studies were retrospectively analyzed for the maximum diameter. The mean duration of the follow-up period was 5.8 years. Operative mortality did not occur. There was no difference in complications, such as stroke (G: 2 vs. E: 0, p = 0.30), paraplegia (G: 1 vs. E: 1, p = 0.66), and respiratory failure (G: 2, vs. E: 0, p = 0.30). The difference in preoperative factors was observed, including the intervals between onset and operation (G; 4.9 years vs. E; 1.9 years, p < 0.01), maximum diameter in preoperative CT (G; 59.0 mm vs. E; 50.5 mm, p < 0.001), and maximum false lumen diameter (G; 35.5 mm vs. E; 29.0 mm, p < 0.01). There was no significant difference in the mid- and long-term survival rates (p = 0.49), aorta-related deaths (p = 0.33), and thoracic re-intervention rates (p = 0.34). Postoperative adverse events occurred in Group E: four cases of retrospective type A aortic dissection, two cases of aorto-bronchial fistula, and one case of aorto-esophagus fistula. Aorta-related death and re-intervention rates crossed over in both groups after seven years postoperatively. Although endovascular repair of CTBAD is less invasive, the rate of freedom from re-intervention was unsatisfactory. Some fatal complications were observed in the endovascular group, and the mid- and long-term outcomes were reversed compared with those in the OSR group. Although OSR is an invasive procedure, it could be performed safely without perioperative complications. OSR has more feasible mid- and long-term outcomes.

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来源期刊
Heart and Vessels
Heart and Vessels 医学-外周血管病
CiteScore
3.10
自引率
13.30%
发文量
211
审稿时长
2 months
期刊介绍: Heart and Vessels is an English-language journal that provides a forum of original ideas, excellent methods, and fascinating techniques on cardiovascular disease fields. All papers submitted for publication are evaluated only with regard to scientific quality and relevance to the heart and vessels. Contributions from those engaged in practical medicine, as well as from those involved in basic research, are welcomed.
期刊最新文献
Chest-CT-based radiomics feature of epicardial adipose tissue for screening coronary atherosclerosis. Persistently low tricuspid annular plane systolic excursion and its prognosis in Japanese hospitalized patients with heart failure with reduced ejection fraction. Acknowledgement to reviewers. Transaortic and transfemoral stent deployment approaches related morphological and clinical outcomes after type II hybrid arch repair for type A aortic dissection. 5-Aminolevulinic acid combined with ferrous iron ameliorates myocardial ischemia/reperfusion injury by increasing heme oxygenase-1.
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