慢性肺病患者右心房自发性破裂导致猝死:病例报告与回顾。

Jitendra S. Tomar, Bajrang K Singh, Surya Prakash, Pramendra S Thakur
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摘要

右心房破裂导致心包积血而猝死的情况并不常见,尤其是在没有任何外伤或心房壁病变迹象的情况下自发发生。死者是一名 32 岁的男子。午餐时间,他开始出现不安、眩晕和昏迷症状,并在 45 分钟内被送往医院。急诊室无法记录他的生命体征,经过心肺复苏后,他被宣布死亡。尸检发现,他有孤立的右心房破裂,心壁没有任何病变。右心房的心壁最薄弱,经常因腔内压力升高而发生自发性破裂。在心包腔内发现了液态血液和凝血块。与心室破裂时血液进入心包的速度相比,心房破裂时血液进入心包的速度要慢得多,因此低心房压力会促进血凝块的形成。发现有慢性肺部疾病的证据,这也是心腔内压升高的原因。即使没有外伤或心肌梗塞病史,贝克三联征--颈静脉充血、心音低沉和低血压--也应提醒急诊室工作人员注意心脏填塞的可能性,因为在极不可能的情况下,可能会发生自发性心壁破裂。
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Sudden death due to spontaneous rupture of the right atrium in the setting of chronic lung disease: A case report and review.
Sudden death from haemopericardium as a result of a right atrial rupture is uncommon, most particularly when this occurs spontaneously without any prior trauma or evidence of atrial wall pathology. The deceased was a 32-year-old man. At lunchtime his symptoms of unease, giddiness and unconsciousness began and within 45 minutes he arrived at the hospital. His vitals could not be recorded in the emergency department, and after CPR, he was pronounced dead. At autopsy, an isolated right atrial rupture, without any disease of the heart wall, was discovered. The right atrium has the weakest wall and is frequently the site of spontaneous rupture brought on by increased intraluminal pressure. Both liquid blood and clotted blood were found in the pericardial cavity. Low atrial pressure encourages clot formation because it causes considerably slower blood entry into the pericardium at the time of atrial rupture compared with entry at the time of ventricular rupture. Evidence of chronic lung disease was found which explains the raised intraluminal pressure of the heart chambers. Even with no history of trauma or myocardial infarction, the Beck triad - an engorged neck vein, a muffled heart sound, and low blood pressure - should alert the emergency room staff to the possibility of cardiac tamponade because, in a very unlikely scenario, spontaneous cardial wall rupture might occur.
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