Xing Wang, Dingke Wen, Fan Xia, Mei Fang, Jun Zheng, Chao You, Lu Ma
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引用次数: 0
摘要
现有研究表明,在蛛网膜下腔出血(SAH)后的亚急性阶段,白质损伤有可能得到修复。然而,由于中枢神经系统的细胞异质性,阐明脑细胞亚群在 SAH 发病的急性期和亚急性期的作用仍具有挑战性。本研究对 SAH 模型小鼠进行了单细胞 RNA 测序,以划分不同的细胞群。通过基因组富集分析(Gene Set Enrichment Analysis)确定了涉及的通路,并使用 R 软件中的 CellChat 软件包探索了细胞间的相互作用。研究结果的验证采用了综合方法,包括磁共振成像、免疫荧光双重染色和 Western 印迹分析。这项研究确定了具有细胞类型特异性基因表达模式的十大脑集群。值得注意的是,在 SAH 后的亚急性阶段,我们观察到修复性小胶质细胞在白质丰富区域的浸润和克隆扩增。此外,我们还发现小胶质细胞相关多养蛋白(PTN)在介导 SAH 模型小鼠少突胶质前体细胞(OPCs)的调控中发挥作用,这与 mTOR 信号通路的激活有关。这些发现强调了小胶质细胞-少突胶质细胞(OPC)之间的相互作用可能会通过 PTN 途径发生,从而在 SAH 后的亚急性阶段对白质修复起到潜在的促进作用。我们的分析揭示了 SAH 后急性期和亚急性期的精确转录变化,为了解 SAH 的机制和开发针对特定细胞亚型的药物提供了启示。
Single-Cell Transcriptomics Revealed White Matter Repair Following Subarachnoid Hemorrhage.
Existing research indicates the potential for white matter injury repair during the subacute phase following subarachnoid hemorrhage (SAH). However, elucidating the role of brain cell subpopulations in the acute and subacute phases of SAH pathogenesis remains challenging due to the cellular heterogeneity of the central nervous system. In this study, single-cell RNA sequencing was conducted on SAH model mice to delineate distinct cell populations. Gene Set Enrichment Analysis was performed to identify involved pathways, and cellular interactions were explored using the CellChat package in R software. Validation of the findings involved a comprehensive approach, including magnetic resonance imaging, immunofluorescence double staining, and Western blot analyses. This study identified ten major brain clusters with cell type-specific gene expression patterns. Notably, we observed infiltration and clonal expansion of reparative microglia in white matter-enriched regions during the subacute stage after SAH. Additionally, microglia-associated pleiotrophin (PTN) was identified as having a role in mediating the regulation of oligodendrocyte precursor cells (OPCs) in SAH model mice, implicating the activation of the mTOR signaling pathway. These findings emphasize the vital role of microglia-OPC interactions might occur via the PTN pathway, potentially contributing to white matter repair during the subacute phase after SAH. Our analysis revealed precise transcriptional changes in the acute and subacute phases after SAH, offering insights into the mechanism of SAH and for the development of drugs that target-specific cell subtypes.
期刊介绍:
Translational Stroke Research covers basic, translational, and clinical studies. The Journal emphasizes novel approaches to help both to understand clinical phenomenon through basic science tools, and to translate basic science discoveries into the development of new strategies for the prevention, assessment, treatment, and enhancement of central nervous system repair after stroke and other forms of neurotrauma.
Translational Stroke Research focuses on translational research and is relevant to both basic scientists and physicians, including but not restricted to neuroscientists, vascular biologists, neurologists, neuroimagers, and neurosurgeons.