作者更正:ADH1C 通过 ADH1C/PHGDH /PSAT1/serine 代谢途径抑制结直肠癌的进展。

IF 6.9 1区 医学 Q1 CHEMISTRY, MULTIDISCIPLINARY Acta Pharmacologica Sinica Pub Date : 2024-11-01 DOI:10.1038/s41401-024-01319-3
Sha Li, Hong Yang, Wan Li, Jin-Yi Liu, Li-Wen Ren, Yi-Hui Yang, Bin-Bin Ge, Yi-Zhi Zhang, Wei-Qi Fu, Xiang-Jin Zheng, Guan-Hua Du, Jin-Hua Wang
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Author Correction: ADH1C inhibits progression of colorectal cancer through the ADH1C/PHGDH /PSAT1/serine metabolic pathway.
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来源期刊
Acta Pharmacologica Sinica
Acta Pharmacologica Sinica 医学-化学综合
CiteScore
15.10
自引率
2.40%
发文量
4365
审稿时长
2 months
期刊介绍: APS (Acta Pharmacologica Sinica) welcomes submissions from diverse areas of pharmacology and the life sciences. While we encourage contributions across a broad spectrum, topics of particular interest include, but are not limited to: anticancer pharmacology, cardiovascular and pulmonary pharmacology, clinical pharmacology, drug discovery, gastrointestinal and hepatic pharmacology, genitourinary, renal, and endocrine pharmacology, immunopharmacology and inflammation, molecular and cellular pharmacology, neuropharmacology, pharmaceutics, and pharmacokinetics. Join us in sharing your research and insights in pharmacology and the life sciences.
期刊最新文献
Dependence of NPPS creates a targetable vulnerability in RAS-mutant cancers. Smad transcription factors as mediators of 7 transmembrane G protein-coupled receptor signalling. Intestinal human carboxylesterase 2 (CES2) expression rescues drug metabolism and most metabolic syndrome phenotypes in global Ces2 cluster knockout mice. Luteolin ameliorates chronic stress-induced depressive-like behaviors in mice by promoting the Arginase-1+ microglial phenotype via a PPARγ-dependent mechanism. Author Correction: ADH1C inhibits progression of colorectal cancer through the ADH1C/PHGDH /PSAT1/serine metabolic pathway.
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