CircTOP1 靶向调控 PTBP1 表达促进冠状动脉钙化的进展

IF 3.5 3区 生物学 Q3 CELL BIOLOGY Experimental cell research Pub Date : 2024-07-15 Epub Date: 2024-06-27 DOI:10.1016/j.yexcr.2024.114147
Hao Hu , Shichun Shen , Jiawei Wu , Likun Ma
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引用次数: 0

摘要

冠状动脉钙化(CAC)是心血管疾病发病机制中的一个标志性事件,涉及血管平滑肌细胞(VSMC)向成骨状态的表型转化。尽管有了这种认识,但支配血管平滑肌细胞成骨转换的分子机制仍未完全阐明。在此,我们试图研究环状 RNA(circRNA)在 CAC 中的潜在作用。通过 circRNA-seq 的转录组分析,我们发现 circTOP1 是 CAC 患者的潜在候选 circRNA。此外,我们还观察到,在 CAC 模型中,circTOP1 的过表达会加剧血管钙化。随后的牵引实验显示,circTOP1 与 PTBP1(circTOP1 在 CAC 中的假定靶基因)之间存在相互作用。在体内和体外实验中,我们观察到 circTOP1 和 PTBP1 在 CAC 模型中的高表达,并注意到减少 circTOP1 的表达可有效减少模型小鼠体内的钙盐沉积和矿化结节。此外,体外实验表明,过量表达 PTBP1 可逆转沉默 circTOP1 所导致的信号减弱,从而加剧 VSMC 的成骨转变和钙化。总之,我们的研究结果表明,circTOP1 通过调节 PTBP1 的表达来介导 VSMC 的转分化,从而促进 CAC 的形成。
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CircTOP1 targeted regulation of PTBP1 expression promotes the progression of coronary artery calcification

Coronary artery calcification (CAC) is a hallmark event in the pathogenesis of cardiovascular disease, involving the phenotypic transformation of vascular smooth muscle cells (VSMC) towards an osteogenic state. Despite this understanding, the molecular mechanisms governing the VSMC osteogenic switch remain incompletely elucidated. Here, we sought to examine the potential role of circular RNA (circRNA) in the context of CAC. Through transcriptome analysis of circRNA-seq, we identified circTOP1 as a potential candidate circRNA in individuals with CAC. Furthermore, we observed that overexpression of circTOP1 exacerbated vascular calcification in a CAC model. Subsequent pull-down assays revealed an interaction between circTOP1 and PTBP1, a putative target gene of circTOP1 in the context of CAC. In both in vivo and in vitro experiments, we observed heightened expression of circTOP1 and PTBP1 in the CAC model, and noted that reducing circTOP1 expression effectively reduced calcium salt deposits and mineralized nodules in model mice. Additionally, in vitro experiments demonstrated that overexpression of PTBP1 reversed the weakening of signaling caused by silencing circTOP1, thereby exacerbating the osteogenic transition and calcification of VSMC. Collectively, our findings suggested that circTOP1 promotes CAC by modulating PTBP1 expression to mediate VSMC transdifferentiation.

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来源期刊
Experimental cell research
Experimental cell research 医学-细胞生物学
CiteScore
7.20
自引率
0.00%
发文量
295
审稿时长
30 days
期刊介绍: Our scope includes but is not limited to areas such as: Chromosome biology; Chromatin and epigenetics; DNA repair; Gene regulation; Nuclear import-export; RNA processing; Non-coding RNAs; Organelle biology; The cytoskeleton; Intracellular trafficking; Cell-cell and cell-matrix interactions; Cell motility and migration; Cell proliferation; Cellular differentiation; Signal transduction; Programmed cell death.
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