P2X7 受体拮抗剂 A740003 对实验性青光眼视网膜神经节细胞的神经保护作用

IF 1.6 4区 医学 Q4 NEUROSCIENCES Neuroreport Pub Date : 2024-09-04 Epub Date: 2024-07-08 DOI:10.1097/WNR.0000000000002071
Yan Zhu, Shu-Ying Li, Lu-Jia Zhang, Bo Lei, Yong-Chen Wang, Zhongfeng Wang
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引用次数: 0

摘要

本研究旨在探讨P2X7受体拮抗剂A740003对慢性眼内高压(COH)实验性青光眼小鼠模型视网膜神经节细胞(RGC)的神经保护作用。生物信息学分析了青光眼相关基因。利用Western印迹、实时荧光定量PCR和免疫荧光染色技术探讨了A740003对COH视网膜中RGCs神经保护作用的机制。生物信息学分析表明,氧化应激、神经炎症和细胞凋亡与青光眼的发病机制高度相关。在 COH 视网膜中,眼压升高会显著增加小胶质细胞活化标志物易位体蛋白的水平,而在玻璃体内预先注射 A740003 可以逆转这一现象。A740003 还能抑制 COH 视网膜中促炎细胞因子白细胞介素(IL)1β 和肿瘤坏死因子 α mRNA 水平的升高。此外,虽然 COH 视网膜中抗炎细胞因子 IL-4 和 IL-10 的 mRNA 水平保持不变,但服用 A740003 可提高其水平。在注射或不注射 A740003 的 COH 视网膜中,Bax 和裂解的 caspase-3 的 mRNA 和蛋白水平均升高,而 A740003 可部分逆转这种升高,Bcl-2 的水平则保持不变。此外,A740003还部分缓解了COH小鼠Brn-3a阳性RGC数量的减少。A740003可以通过抑制小胶质细胞的活化、减轻视网膜炎症反应、减少RGCs的凋亡以及提高COH实验性青光眼中RGCs的存活率,从而对RGCs起到神经保护作用。
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Neuroprotection of the P2X7 receptor antagonist A740003 on retinal ganglion cells in experimental glaucoma.

The aim of this study was to explore the neuroprotective effects of the P2X7 receptor antagonist A740003 on retinal ganglion cells (RGCs) in chronic intraocular hypertension (COH) experimental glaucoma mouse model. Bioinformatics was used to analyze the glaucoma-related genes. Western blot, real-time fluorescence quantitative PCR, and immunofluorescence staining techniques were employed to explore the mechanisms underlying the neuroprotective effects of A740003 on RGCs in COH retinas. Bioinformatic analysis revealed that oxidative stress, neuroinflammation, and cell apoptosis were highly related to the pathogenesis of glaucoma. In COH retinas, intraocular pressure elevation significantly increased the levels of translocator protein, a marker of microglial activation, which could be reversed by intravitreal preinjection of A740003. A740003 also suppressed the increased mRNA levels of proinflammatory cytokines interleukin (IL) 1β and tumor necrosis factor α in COH retinas. In addition, although the mRNA levels of anti-inflammatory cytokine IL-4 and IL-10 were kept unchanged in COH retinas, administration of A740003 could increase their levels. The mRNA and protein levels of Bax and cleaved caspase-3 were increased in COH retinas, which could be partially reversed by A740003, while the levels of Bcl-2 kept unchanged in COH retinas with or without the injections of A740003. Furthermore, A740003 partially attenuated the reduction in the numbers of Brn-3a-positive RGCs in COH mice. A740003 could provide neuroprotective roles on RGCs by inhibiting the microglia activation, attenuating the retinal inflammatory response, reducing the apoptosis of RGCs, and enhancing the survival of RGCs in COH experimental glaucoma.

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来源期刊
Neuroreport
Neuroreport 医学-神经科学
CiteScore
3.20
自引率
0.00%
发文量
150
审稿时长
1 months
期刊介绍: NeuroReport is a channel for rapid communication of new findings in neuroscience. It is a forum for the publication of short but complete reports of important studies that require very fast publication. Papers are accepted on the basis of the novelty of their finding, on their significance for neuroscience and on a clear need for rapid publication. Preliminary communications are not suitable for the Journal. Submitted articles undergo a preliminary review by the editor. Some articles may be returned to authors without further consideration. Those being considered for publication will undergo further assessment and peer-review by the editors and those invited to do so from a reviewer pool. The core interest of the Journal is on studies that cast light on how the brain (and the whole of the nervous system) works. We aim to give authors a decision on their submission within 2-5 weeks, and all accepted articles appear in the next issue to press.
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