木犀草素能缓解D-半乳糖诱导的大鼠脑衰老:SIRT1 介导的神经保护。

IF 3.7 3区 医学 Q2 BIOCHEMISTRY & MOLECULAR BIOLOGY Neurochemical Research Pub Date : 2024-10-01 Epub Date: 2024-07-11 DOI:10.1007/s11064-024-04203-y
Reham L Younis, Rehab M El-Gohary, Asmaa A Ghalwash, Islam Ibrahim Hegab, Maram M Ghabrial, Azza M Aboshanady, Raghad A Mostafa, Alaa H Abd El-Azeem, Eman E Farghal, Asmaa A E Belal, Haidy Khattab
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引用次数: 0

摘要

叶黄素是一种存在于多种植物中的重要天然多酚。大量研究证实了叶黄素在神经退行性疾病中的保护作用,但对其在 D-半乳糖(D-gal)诱导的脑衰老中的治疗作用的研究仍然缺乏。本研究探讨了叶黄素对 D-gal 诱导的脑老化的潜在神经保护作用。研究人员将 40 只大鼠随机分为四组:对照组、木犀草素组、D-gal 组和木犀草素注射 D-gal 组。所有组均接受行为、胆碱能功能和海马线粒体呼吸评估。检测海马氧化、神经炎症、衰老和凋亡指标。评估了 SIRT1、BDNF 和 RAGE 的基因表达。还进行了海马组织病理学研究,以及 GFAP 和 Ki67 免疫反应。我们的研究结果表明,叶黄素能有效缓解 D-gal 诱导的认知障碍,并逆转胆碱能异常。此外,叶黄素还能显著减轻D-gal诱导的海马氧化应激、线粒体功能障碍、神经炎症和衰老,并能显著减少神经元凋亡和上调海马SIRT1 mRNA的表达。总之,我们的研究结果表明,叶黄素通过抗氧化、抗衰老、抗炎症和抗凋亡作用,可减轻D-gal诱发的脑衰老,改善线粒体功能,促进老龄大鼠海马神经再生,这可能是由于SIRT1的上调所致。木犀草素可能是治疗脑衰老相关异常的一种很有前景的方法。
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Luteolin Mitigates D-Galactose-Induced Brain Ageing in Rats: SIRT1-Mediated Neuroprotection.

Luteolin is an essential natural polyphenol found in a variety of plants. Numerous studies have supported its protective role in neurodegenerative diseases, yet the research for its therapeutic utility in D-galactose (D-gal)-induced brain ageing is still lacking. In this study, the potential neuroprotective impact of luteolin against D-gal-induced brain ageing was explored. Forty rats were randomly divided into four groups: control, luteolin, D-gal, and luteolin-administered D-gal groups. All groups were subjected to behavioural, cholinergic function, and hippocampal mitochondrial respiration assessments. Hippocampal oxidative, neuro-inflammatory, senescence and apoptotic indicators were detected. Gene expressions of SIRT1, BDNF, and RAGE were assessed. Hippocampal histopathological studies, along with GFAP and Ki67 immunoreactivity, were performed. Our results demonstrated that luteolin effectively alleviated D-gal-induced cognitive impairment and reversed cholinergic abnormalities. Furthermore, luteolin administration substantially mitigated hippocampus oxidative stress, mitochondrial dysfunction, neuro-inflammation, and senescence triggered by D-gal. Additionally, luteolin treatment considerably attenuated neuronal apoptosis and upregulated hippocampal SIRT1 mRNA expression. In conclusion, our findings revealed that luteolin administration attenuated D-gal-evoked brain senescence, improving mitochondrial function and enhancing hippocampal neuroregeneration in an ageing rat model through its antioxidant, senolytic, anti-inflammatory, and anti-apoptotic impacts, possibly due to upregulation of SIRT1. Luteolin could be a promising therapeutic modality for brain aging-associated abnormalities.

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来源期刊
Neurochemical Research
Neurochemical Research 医学-神经科学
CiteScore
7.70
自引率
2.30%
发文量
320
审稿时长
6 months
期刊介绍: Neurochemical Research is devoted to the rapid publication of studies that use neurochemical methodology in research on nervous system structure and function. The journal publishes original reports of experimental and clinical research results, perceptive reviews of significant problem areas in the neurosciences, brief comments of a methodological or interpretive nature, and research summaries conducted by leading scientists whose works are not readily available in English.
期刊最新文献
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